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Plasmacytoid dendritic cells, which overproduce IFN-α, may play a crucial role in starting alopecia areata, an autoimmune disease causing hair loss.

Hair follicles prevent NK cell attacks to avoid hair loss.

Autoimmune diseases need combined therapy of tolerance induction and immunosuppression.

Type 1/17 inflammation in psoriasis increases skin cell growth due to a molecule that could be a new treatment target.

iNKT cells can help prevent and treat alopecia areata by promoting hair regrowth.

Researchers developed a 3D skin model with its own immune and blood vessel cells to better understand skin health and disease.

Hair follicles regulate immune cells in the skin, and disrupting certain pathways can lead to skin issues and infections.

A specific group of slow-growing stem cells marked by Thy1 is crucial for skin maintenance and healing in mice.

Patients with severe active alopecia areata have lower CD200 expression and an imbalance in their immune system.

Scientists found a new type of skin cell that could help with skin repair and these cells work better with a certain protein.

Both Th1 and Th2 immune responses are increased in alopecia areata, with Th2 response more strongly linked to how severe the disease is.

Hair stem cells produce a protein called COL17A1 that plays a key role in their development and is linked to hair thinning and baldness.

Higher levels of IL-1a and IL-1RA were found in severe alopecia areata cases.

Activin increases skin tumor formation, skin Tregs help hair growth, lymph-node removal doesn't improve melanoma survival, cells can revert to stem cells in wound healing, and skin bacteria produce peptides that may treat infections.

Skin fat plays a key role in immune defense and healing beyond just storing energy.

The document concludes that DAB389-IL2 is promising for treating refractory cutaneous T-cell lymphoma, but more research is needed on its effectiveness and side effect management.

Dithranol reduces inflammation in alopecia areata by lowering certain immune responses.

CD8+ T cells re-expressing CD45RA may predict treatment resistance in severe alopecia areata.

Certain immune cells help hair growth by regulating iron in the skin.

Ancient immune and signaling pathways still regulate blood cell development.

Giving immune serum from vaccinated mice to mice without T cells prevents infection and tumor growth.

Innate lymphoid cells type 1 may contribute to alopecia areata by damaging hair follicles.

Hair follicles help attract immune cells to minor skin injuries.

People on immune-modifying skin disease treatments may have a weaker antibody response to COVID-19 vaccines but often improve after the second dose.

Vitiligo is caused by the immune system attacking skin pigment cells.

BST2 protein and certain T cells increase in early alopecia areata.

Immunomodulatory therapies are effective for treating cutaneous lymphoma, particularly in early stages.

A parasite molecule can speed up skin healing and reduce scarring.