Search

everythinglearnresearchcommunity

for

Search:↓

Iron deficiency worsens inflammatory skin diseases by disrupting iron balance and increasing inflammation.

TLR3 activation helps improve skin and hair follicle healing in mice.

Keratin 17 is important for skin development and may help define skin cell types.

TCDD disrupts skin stem cells, causing skin issues like chloracne.

Aggressive immunosuppressive treatment improved a woman's severe heart condition linked to autoimmune disease.

IL-15 helps protect hair follicles from immune attacks and encourages hair growth.

Collagen XVII is crucial for skin cell growth and nail health.

Treatment with biologic agents can significantly improve psoriasis symptoms, and blood biomarkers could potentially predict individual patient's response to treatment.

Organ transplant recipients have a higher risk of skin cancer over time, atopic dermatitis skin shows unusual bacterial and fungal patterns, a new tool for measuring hidradenitis suppurativa severity was created, and gene expression changes in male baldness suggest new treatments.

Special immune cells called Tregs can help prevent lung scarring by blocking a specific growth factor.

Double-stranded RNA causes inflammation in hair follicle cells, which may help understand and treat alopecia areata.

TLR3 signaling enhances the immunosuppressive properties of human periodontal ligament stem cells.

TPA strongly increases ODC activity in certain skin cells, potentially aiding tumor growth.

Certain types of T cells are essential for healthy skin and play a role in skin diseases, but more research is needed to improve treatments.

Wnt5a may contribute to psoriasis by reducing skin cell growth, blocking skin cell maturation, and increasing inflammation.

Certain inflammatory factors may increase or decrease the risk of developing PCOS.

ILC1-like cells may contribute to hair loss in alopecia areata.

High DHA levels delay wound healing and worsen skin repair quality.

Acne inversa is an epithelial-driven disease where inflammation is caused by cyst rupture, and treatments should focus on preventing tendril growth for better results.

TCDD changes skin cell growth and keratin production in mice.

ILC1-like cells can independently cause alopecia areata.

IL-27 may help prevent hair loss by creating immune-suppressing cells.

Certain genetic variations in IL-16 may increase the risk of alopecia areata.

Certain IL-18 gene variations may increase the risk of alopecia areata.

Higher levels of IL-1R1 and hsa-miR-19b-3p may help diagnose and predict alopecia areata severity.

BST2 is highly expressed in certain immune cells in alopecia areata, suggesting a role in the disease.

Innate lymphoid cells type 1 may contribute to alopecia areata by damaging hair follicles.

ILC1-like cells can cause alopecia areata by disrupting hair follicle immunity, suggesting a new treatment approach.