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Hair follicles may help teach the immune system to tolerate new self-antigens, but this can sometimes cause hair loss.

Plasmacytoid dendritic cells, which overproduce IFN-α, may play a crucial role in starting alopecia areata, an autoimmune disease causing hair loss.

The document concludes that DAB389-IL2 is promising for treating refractory cutaneous T-cell lymphoma, but more research is needed on its effectiveness and side effect management.

People with atopic dermatitis have more CD4+ T cells that respond to a certain bacterial lipid, which may play a role in the skin condition's inflammation.

CD18-deficient mice developed psoriasis-like skin disease, useful for studying inflammatory skin disorders.

Regulatory T cells protect hair follicle stem cells by maintaining immune privilege in the skin.

The role of γδT-cells in causing alopecia areata remains unclear.

A child with trichothiodystrophy also had autoimmune thyroiditis and anemia, which is a new finding.

IL-13 protein is much higher in the skin of atopic dermatitis patients than in healthy skin.

BST2 protein and certain T cells increase in early alopecia areata.

IL-1 signaling is crucial for hair follicle stem cell growth and wound healing.

Targeting THY1 can improve skin repair and healing.

Mutations in TBX3 cause horses to have more even hair color instead of Dun camouflage.

Different parts of the body have unique immune characteristics in their skin.

ILC1 cells contribute to hair loss in alopecia areata.

Innate lymphoid cells type 1 may contribute to alopecia areata.

Clonally expanded CD8+ T cells cause alopecia areata.

Dermal IgA deposition without symptoms is rare in Dermatitis herpetiformis risk groups.

In alopecia areata, certain immune cells increase and express a protein linked to immune activation.

Severe alopecia areata involves higher levels of certain immune cells, which can be normalized with betamethasone.

Mice lacking steroid 5α-reductase 2 show less aggression and better impulse control.

B2m-free HLA variants may be a new class of HLA important in immune responses and diseases.

Damaging mutations in NFKB2 cause a severe and distinct form of primary immunodeficiency with early-onset and often ACTH-deficiency.

A patient with Ivemark syndrome developed mixed type vitiligo after a hepatitis C infection, showing different treatment responses and immune cell involvement in the skin.

Injecting recipient splenocytes into donors' thymus can prevent graft-versus-host disease.

A protein called lfTSLP is important in causing allergic and other skin diseases and could be a target for treatment.

CD1d expression in scalp skin and hair follicles changes with the hair cycle and may help protect against microbes.