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Hair follicle stem cell transplants can reverse liver cirrhosis by blocking harmful cell activation.

Hedgehog signaling can create new hair follicles but may also cause tumors.

Hair follicle stem cells are a practical and ethical option for nerve repair in regenerative medicine.

CD8+ T cells are involved in alopecia areata and may cause disease relapse.

The assay effectively identifies compounds that affect immune cell activation.

Researchers found a new gene, hacl-1, that is active in mouse hair follicles during hair growth and may be important for hair biology.

Lgr6+ cells are important for tissue repair and could be a target for treating cancer and other diseases.

Expanding CD4+ Tregs can stop hair loss in alopecia areata.

HSD11b1 affects skin nerves and increases non-histaminergic itch.

3D culture better preserves key properties of hair follicle cells for regeneration.

4-O-Methylhonokiol helps protect skin cells from growth-stopping effects of a protein by regulating growth-related pathways.

Overexpressing thrombospondin-1 in mice skin prevents UVB-induced skin damage.

Dying cells can help with faster healing and new hair growth by releasing a growth-promoting molecule.

CCL5 is important for the hair growth potential of human dermal papilla cells.

Transplanted bone marrow cells actively move, form clusters, and grow after transplantation.

Higher PD-1 levels mean fewer CD8+ T cells in alopecia areata hair follicles.

Researchers created a cell model to study hair growth and test hair-growth drugs.

Higher thyroid antibodies in Hashimoto's thyroiditis are linked to more inflammation and symptoms.

Exosomes from skin cells can boost hair growth by stimulating a gene called LEF1.

Special immune cells called Tregs can help prevent lung scarring by blocking a specific growth factor.

Trypsin slows hair growth and affects color by causing cell death in hair follicles.

Thyroid hormone receptors are essential for hair growth and wound healing.

Adipose-derived stem cells can be used to create cells that help grow new hair.

Different γδ T cell types have unique roles in causing alopecia areata.

TTD patients don't have a higher skin cancer risk because their main issue is with transcription, not DNA repair.

The mouse model showed defects in adult stem cell maintenance related to Hutchinson-Gilford progeria syndrome.

The conclusion is that the dermal papilla is crucial for hair follicle regrowth, and hair follicles undergo significant structural changes in addition to cell division during regeneration.

A virus protein can activate a pathway that may lead to abnormal hair follicle development.