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Women with Polycystic Ovary Syndrome (PCOS) have lower levels of a substance called DIAPH1 in their blood, which is linked to changes in sugar metabolism and insulin resistance.

New mutations in the hairless gene may cause hair loss and affect bone development.

Changing the amount of PLC5 in Arabidopsis affects root growth and drought resistance, with less PLC5 slowing root growth and more PLC5 improving drought tolerance but hindering root hair growth.

Mouse hair follicle stem cells have a flexible chromatin state that supports skin health and hair growth.

Mealworm protein helps fat cell development and may aid in metabolic health and hair growth.

Targeting THY1 can improve skin repair and healing.

Higher thyroid hormone levels may be linked to certain types of polycystic ovary syndrome.

Root hairs help Arabidopsis plants absorb more phosphorus when it's scarce.

Quiescent cells have increased mitochondrial activity and ECM gene expression, but reduced glycolysis.

Prenatal diagnosis of trichothiodystrophy is possible in the second trimester using fetal eyebrow biopsy, with sulfur content analysis being more reliable.

Arabidopsis thaliana root hairs efficiently acquire phosphorus in low-phosphorus conditions.

New gene mutations linked to skin conditions were found, bacteria and chemicals may worsen acne, a dog mutation could exist in humans, virus-like elements might be involved in psoriasis, and a vitamin D3 treatment doesn't prevent chemotherapy-related hair loss.

848 genes related to fat and metabolism are less active in people with Central Centrifugal Cicatricial Alopecia.

The MTR gene affects wool quality and production in Chinese Merino sheep.

The type of PCOS a woman has doesn't strongly predict her skin or metabolic symptoms; obesity is a more important factor.

L-threonate may help prevent balding by blocking a key hair loss factor.

FTase and GGTase-I are essential for skin keratinocyte health.

Retinoids or their analogs could treat skin pigmentation disorders like melasma and vitiligo.

Scientists made a mouse that can be made to lose hair and then grow it back.

Malnourished people have weaker hair that is easier to pluck.

CRISPR gene editing reduces harmful molecules in cells from Emery–Dreifuss Muscular Dystrophy patients.

Increased LC3 gene expression may be linked to premature graying of hair.

The document concludes that mouse models are crucial for studying hair biology and that all mutant mice may have hair growth abnormalities that require detailed analysis to identify.

Hairless protein and putrescine regulate each other, affecting hair growth and skin balance.

SCD1 is crucial for skin health and overall energy balance.

Phosphorylation controls TFEB's location in the cell, affecting cell metabolism and stress response.

Overexpressing 14-3-3σ in mice skin reduces cell growth and hair density.

The 736T>A mutation in the LIPH gene is common in Japanese people with autosomal recessive woolly hair.

The protein STAT3 slows down cell growth by blocking the FST gene, which affects hair development in sheep.

Glutamine metabolism affects hair stem cell maintenance and their ability to change back to stem cells.