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The protein CTCF is essential for skin development, maintaining hair follicles, and preventing inflammation.

Type II keratins are uniquely phosphorylated during stress and mitosis, affecting their structure and function.

The C-terminal tail of AHF/trichohyalin is essential for organizing keratin filaments in keratinocytes.

Tcf3 helps cells move and heal wounds by controlling lipocalin 2.

Cathepsin E is crucial for normal skin cell differentiation and development.

Canine hypothyroidism often causes skin issues like hair loss.

Desmoglein 4 is controlled by specific proteins that affect hair growth.

CD4+ skin cells may be precursors to basal cell carcinoma.

Different keratins have unique expression patterns in mouse skin cells.

BST2 is highly expressed in certain immune cells in alopecia areata, suggesting a role in the disease.

A specific hair protein variant increases the spread of breast cancer and is linked to worse survival rates.

TGF-beta 1 and IGF-II mRNA have specific patterns in pig subcutaneous tissue, affecting fat and muscle development.

The mTurq2-Col4a1 mouse model shows how the basement membrane develops in live mammals.

Spt4 and Spt6 are essential for fat cell development.

TAK-279 effectively reduces psoriasis symptoms and is safe.

Changes to the Foxp3 protein affect how well regulatory T cells can control the immune system, which could help treat immune diseases and cancer.

Genetic testing can diagnose hair loss linked to DSG4 gene variants.

Lower TGFß1 levels help stem cells become beige fat cells.

A new DSG4 gene mutation causes hair defects in a young girl.

4-O-Methylhonokiol helps protect skin cells from growth-stopping effects of a protein by regulating growth-related pathways.

Thyroid function affects skin health, with a complex interaction between the two.

A new mutation in the ST14 gene broadens the understanding of ichthyosis-hypotrichosis syndrome.

Researchers converted human embryonic stem cells into trophoblast stem cells using specific transcription factors.

Mutations in the DSG4 gene cause fragile, sparse hair in humans, mice, and rats.

Vitamin D receptor can control the hairless gene linked to hair loss even without vitamin D.

A specific group of early-stage melanocytes is reduced in vitiligo-affected skin, which may explain treatment resistance.