Search

everythinglearnresearchcommunity

for

Search:↓

Overexpressing PKCα in mice skin increases inflammation but doesn't affect tumor growth.

Regulatory T cells are essential for normal and improved wound healing in mice.

Laminin-511 is essential for proper melanocyte movement and development in mice.

Targeting NCoR1 can help treat heart enlargement and dysfunction.

The assay effectively identifies compounds that affect immune cell activation.

A mutation in mice causes hair loss and skin issues due to a defect in a gene affecting cell adhesion.

Blocking the CXCR3 receptor reduces T cell accumulation in the skin and prevents hair loss in mice.

Overexpressing 14-3-3σ in mice skin reduces cell growth and hair density.

MC-DNA vector-based gene therapy can temporarily treat CBS deficiency in mice.

Overexpressing SSAT in mice makes them highly sensitive to polyamine analogues, causing liver damage and high mortality.

Cupric chloride treatment corrected abnormal Purkinje cell development in brindled mice.

Hoxc13 gene is essential for hair, nail, and papilla development.

The scraggly mutation causes hair loss and skin defects in mice.

A small change in the TRPS1 gene leads to a less severe form of a syndrome affecting hair, nose, and finger development.

Deleting part of a gene in mice causes wavy hair and high pup loss.

Tet2 and Tet3 enzymes are important for controlling hair growth and shape by affecting gene activity and DNA structure in hair follicles.

The C3H/HeJ mouse model is useful for studying and testing treatments for alopecia areata.

The boy's severe skin disorder is caused by two new mutations in his TGM1 gene.

Loss of the p53 gene alone causes tumors, and losing both p53 and Rb genes speeds up aggressive skin cancer.

Some mutant mice have hair with abnormal cross-linking, mainly in the cuticle, not affecting other hair parts.

Giving immune serum from vaccinated mice to mice without T cells prevents infection and tumor growth.

Keratin 16 delays skin maturation and affects skin and hair development in mice.

Overexpression of LRIG3 in skin causes hair loss.

Mice without the vitamin D receptor gene lose hair due to disrupted hair follicle cycles.

Male-to-female skin grafts in mice are rejected due to sex-linked antigens.

Overexpression of certain genes can shorten hair by disrupting the hair-growth cycle.

IL-18 signaling helps mature Tregs move into the thymus.

A litter of cats had a hair condition similar to a mouse mutation, leading to hair loss and abnormal hair and skin.

Certain mutations in the KLHL24 gene cause a skin disorder by breaking down an important skin protein.