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Pigs without the Hairless gene showed skin and thymus changes, useful for studying human hair disorders.

Li2CO3 improved skin disease in a mouse model of Focal Dermal Hypoplasia without toxicity.

Lack of TrkC receptor delays hair follicle development.

A KLK5 inhibitor effectively improved skin symptoms in a mouse model of Netherton Syndrome.

K6/ODC transgenic mice are effective for quickly identifying cancer-causing chemicals.

Researchers created a new mouse model for studying Citrullinemia Type I and similar conditions, showing symptoms and treatment responses like those in humans.

The HtrA1L364P mutation causes brain dysfunction and blood vessel damage.

A new mouse model for vitiligo helps study immune responses and potential treatments.

Deleting the CD271 gene in mouse skin cells leads to disorganized skin and increased hair growth, suggesting CD271 is important for skin health.

Introducing the rat OTC gene normalized hair growth in SPF-ASH mice.

ING5 is crucial for stem cell maintenance and preventing certain cancers.

A gene mutation causes curly hair and hair loss in rats.

A gene mutation causes lanceolate hair in rats by disrupting hair shaft integrity.

New mouse models help study melanocytic cells for melanoma research.

TSC2 is crucial for proper hair follicle development and patterning.

TAF4 is important for skin cell growth and helps prevent skin cancer in mice.

Mouse models help target specific genes in lymphatic cells for research.

The Dfl mutation in mice causes poor sebaceous gland function and complete hair loss.

RNase L suppresses regeneration in mammals.

The study concludes that a genetic mutation in TFM mice leads to reduced androgen receptor activity, affecting the body's response to male hormones.

Removing Ctip2 in skin cells causes skin inflammation similar to atopic dermatitis.

The HCR gene contributes to psoriasis risk.

Overexpressing COX-2 in mice skin reduces skin tumor development.

A position effect on the TRPS1 gene causes excessive hair growth in humans and mice.

Deleting the MAD2L1 gene is tolerated in certain mouse cancer models.

Tabby mutations in mice affect hair follicle development and help study genetic mapping and certain medical conditions.

Melanocyte pathology requires keratinocyte hyperplasia and regulation dysfunction.

Mutations in specific keratin genes cause improper hair structure in mice due to faulty keratin protein assembly.

Mutations in β1 integrins cause embryonic death but have milder effects on skin.

Removing GRK2 in skin cells causes hair loss similar to immune-related alopecia.