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Tet2 and Tet3 enzymes are important for controlling hair growth and shape by affecting gene activity and DNA structure in hair follicles.

SkQ1 antioxidant improved health and lifespan in mice.

Rhino mice show significant meibomian gland changes, making them a potential model for studying gland disorders.

Suppressing ODC activity reduces tumor growth in hair follicles.

Blocking a specific receptor slows down hair loss in mice.

Neurotrophin-4 increases calcium current in specific mouse neurons through the PI3K pathway.

A new mouse model for vitiligo helps study immune responses and potential treatments.

ODC transgenic mice can model human hair loss with skin lesions.

miRNAs in the Dlk1-Gtl2 region may improve lamb fur quality.

Mutations in TBX3 cause horses to have more even hair color instead of Dun camouflage.

Keratin 6a is important for quick wound healing from hair follicles.

Rac1 is crucial for normal hair structure and pigmentation.

A new method can detect mutations in mice by observing changes in hair follicle cells.

The tumor suppressor gene FLCN affects mitochondrial function and energy use in cells.

Copper supplements during pregnancy improve survival and development in mutant mice.

Tet2 and Tet3 enzymes are essential for controlling hair growth by affecting DNA demethylation and gene expression in mice.

Removing GRK2 in skin cells causes hair loss similar to immune-related alopecia.

Copper treatments increase copper in all tissues, but brindled female mice accumulate much more copper in their kidneys without clinical effects, unlike brindled male mice where brain copper deficiency is clinically significant.

Hereditary factors cause hair loss in mice by affecting skin and hair follicle structure.

The transgene likely activated an oncogene or interrupted a tumor suppressor gene, causing melanoma in mice.

NKIRAS2 can suppress certain skin tumors but its effect on cancer varies with context and expression level.

A new keratin gene was found in mice, explaining hair growth.

Mice without the vitamin D receptor gene lose hair due to disrupted hair follicle cycles.

The Foxn1(-/-) nude mouse shows disrupted and expanded skin stem cell areas due to high Lhx2 levels.

WWP2 is crucial for tooth development in mice.

Disrupting the Flcn gene in mice causes early kidney cysts and tumors, which can be treated with rapamycin.

Not having the gene PLAAT3 leads to fat loss, high insulin resistance, and abnormal fat levels in the blood due to a disruption in fat cell development and function.