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A rare skin reaction from cancer treatment was successfully managed with topical treatments and antihistamines.

Activin increases skin tumor formation, skin Tregs help hair growth, lymph-node removal doesn't improve melanoma survival, cells can revert to stem cells in wound healing, and skin bacteria produce peptides that may treat infections.

Scientists found key proteins and genes that affect skin and hair health, and identified potential new treatments for hair loss, skin disorders, and wound healing.

A new genetic variant causes BRESHECK syndrome by disrupting cell growth and stress response.

PDCD4 is important for controlling skin cell growth and healing.

Increased ODC expression makes normally tumor-resistant mice more prone to tumor development.

Activating c-Myc in skin causes rapid cell growth and changes, but these effects are reversible.

The ABI1 gene contributes to prostate cancer progression and treatment resistance.

Higher TGF-β signaling may increase skin cancer risk in organ transplant recipients.

Activating TERT in mice skin boosts hair growth by waking up hair follicle stem cells.

Vitamin A treatment reduced abnormal cell growth and improved skin conditions in rats with tumors.

The L457(3.43)R mutation in the human lutropin receptor causes increased activity and hormone insensitivity, leading to precocious puberty.

Cepharanthine can potentially treat gastric cancer by stopping tumor growth.

GLI2 activates GLI1, promoting skin tumor growth and hair development.

Hsc70 protein may influence hair growth by responding to androgens.

CCC1 is crucial for pH balance in plant cells, affecting growth and stress tolerance.

Activating TLR3 boosts autophagy gene expression in skin cells.

Allopregnanolone increases growth and changes gene activity in human brain cancer cells.

Budesonide and N-acetylcysteine reduced tumors and alopecia in mice, regardless of FHIT gene status.

Altering the keratin 17 gene in mice hair follicles caused temporary hair issues, but changes were minimal and short-lived.

Most patients experienced mild to moderate skin problems during a trial for a desmoid tumor treatment.

The mTOR pathway may be involved in the development of hair follicle tumors, with higher activity in malignant tumors.

Progranulin overexpression leads to shorter, thinner hair and increased cell death in mouse hair follicles.

Targeting mitophagy may help treat alopecia areata by reducing inflammasome activation.

EGFR deficiency in skin causes hair follicle issues and inflammation.

ISCK03 stops melanin production in human melanoma cells and lightens skin color in mice and guinea pigs.