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Krt6a-Cre transgenic mice help study gene effects on hair follicle development and tumor suppression.

The mouse model helps study and develop treatments for radiation-induced saliva reduction.

Adding TERT and BMI1 to certain skin cells can improve their ability to create hair follicles in mice.

Erythropoietin overexpression disrupts hair growth and fat formation in mice.

Type XIX Collagen is present in specific skin and hair cells during development.

The system allows precise control of gene expression in mouse skin, useful for studying skin biology.

Overexpression of COX-2 in mice skin causes abnormal skin and hair development.

LGD1069 effectively prevents breast tumors in mice without toxicity.

Overexpressing the MSX-2 gene in mice causes skin and hair growth issues.

Mice with extra PKCδ resist chemical-induced skin cancer but not UV-induced.

Lack of functional CYLD in mice leads to early aging and cancer.

Overexpressing 14-3-3σ in mice skin reduces cell growth and hair density.

New mouse models help study melanocytic cells for melanoma research.

Dormant melanoma cells in mice interact minimally with memory T cells due to a suppressive tumor environment.

Melanocyte pathology requires keratinocyte hyperplasia and regulation dysfunction.

Overexpression of activin A in mice skin causes skin thickening, fibrosis, and improved wound healing.

Overexpressing Dsg3 in mice skin causes excessive cell growth and abnormal skin development.

Too much keratin 16 in mice skin causes abnormal skin thickening and structure.

Overexpressing the glucocorticoid receptor in mice leads to abnormal skin development and reduced inflammation.

Mice with human skin protein K8 had more skin problems and cancer.

Activated erbB-2 in mice skin causes severe skin and hair abnormalities.

The Walleye dermal sarcoma virus cyclin causes excessive skin cell growth in mice.

High levels of human keratin 16 in mice cause skin lesions and abnormal skin development.

Wnt5a overexpression alone doesn't cause psoriasis in mice but affects hair growth.

NAG-1 may help prevent some metabolic issues related to PCOS.

Increasing COX-2 in mouse skin causes bigger sebaceous glands and thinner hair, but stopping COX-2 can reverse hair thinning.

Researchers created a new mouse model, G4, that mimics human PCOS symptoms and links the condition to a specific gene.

Melanoma cells lose their ability to form tumors when placed in a zebrafish embryo environment.