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Researchers developed a mouse model that tracks hair growth using bioluminescence, improving accuracy in studying hair cycles.

Mice with extra human KLK14 had hair and skin problems, including weaker cell bonds and inflammation, linked to Netherton syndrome.

Scientists restored fertility in male mice lacking a key fertility gene by using a modified gene.

A new mouse model helps study melanocyte cells using GFP expression.

SV40 T antigen in hair follicles causes abnormal hair and health issues in mice.

Overexpressing SSAT in mice makes them highly sensitive to polyamine analogues, causing liver damage and high mortality.

Minoxidil boosts hair growth in genetically modified mice.

Overexpressing the human H-ferritin gene in mice causes mild growth delay and temporary hair loss.

Proteolytic enzymes damage hair follicles by detaching stem cells.

Genetically modified sheep with more β-catenin grew more wool without changing the wool's length or thickness.

Scientists made a mouse that can be made to lose hair and then grow it back.

The E2 protein affects gene activity in hair follicles of mice.

Overexpressing ovine β-catenin in mice skin increases hair follicle density and growth.

Proteolytic enzymes damaged hair follicle stem cells in transgenic mice.

The Trichodysplasia spinulosa virus protein can cause abnormal hair growth in mice.

Mice with a changed Hr gene lose and regrow hair due to changes in the gene's activity.

The technology can create transgenic cashmere goats with improved wool quality.

Transgenic sheep cells with spider silk gene were successfully created for future sheep hair expression.

EGF affects hair and skin development.

The gene Endothelin 3 makes mice's fur darker by increasing pigment cells and pigment levels.

The mutation causing Hutchinson-Gilford progeria syndrome leads to severe skin problems and early death in mice.

The TCL1 transgenic mouse model is useful for understanding human B-cell leukemia and testing new treatments.

GFP transgenic mice help study cell origins in skin grafts.

Homozygous K5Cre transgenic mice have wavy hair and faster cancer progression.

Modified β-catenin causes different effects in hair and skin cells, leading to cysts or tumors.

Modified β-catenin can cause different effects in mouse skin cells, leading to cysts or tumors depending on the cell type.

c-Myc activation in mouse skin increases sebaceous gland growth and affects hair follicle development.

Overexpressing follistatin in mice delays wound healing and reduces scar size.

High levels of ODC and a mutant Ha-ras gene cause tumors in mice.

XEDAR deficiency prevents muscle degeneration in EDA-A2 transgenic mice.