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NKIRAS2 can suppress certain skin tumors but its effect on cancer varies with context and expression level.

Overexpression of the HE4 gene in mice causes eye inflammation and cloudiness.

HAP stem cells in hair follicles could help repair nerves and spinal cords.

Activating c-Myc in skin causes rapid cell growth and changes, but these effects are reversible.

Nestin is found in hair follicle progenitor cells, linking them to neural stem cells.

Overexpressing GLI-1 in mice skin can cause tumors like human basal cell carcinomas.

Ectodysplasin-A1 is crucial for developing hair, teeth, and glands.

Overexpression of Hoxc13 in hair cells causes hair loss and skin issues.

Ornithine decarboxylase is crucial for hair growth regulation in mice.

The nude gene causes skin cell overgrowth and improper development, leading to hair and urinary issues.

Blocking BMP signaling causes hair loss and disrupts hair growth cycles.

Bcl-2 overexpression protects against UVB damage but worsens hair loss from chemotherapy.

Overexpression of the glucocorticoid receptor in mice causes developmental defects similar to ectodermal dysplasia.

Betacellulin helps blood vessel growth in wounds but delays hair growth.

Increasing neurotrophin 4 in skin boosts nerve endings but not sensory neuron count.

Protein kinase C epsilon may increase skin cancer risk by affecting nearby cells.

HPV16 oncogenes disrupt the normal activity of hair follicle stem cells.

Gasdermin A3 overexpression in skin causes inflammation and hair loss.

The assay effectively identifies compounds that affect immune cell activation.

Suppressing eIF4E can prevent hair loss from chemotherapy.

Progranulin overexpression leads to shorter, thinner hair and increased cell death in mouse hair follicles.

Protein kinase C epsilon may promote skin cancer development after UV exposure by affecting nearby cells.

Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

Profilin1 speeds up wound healing.

Activating β-catenin signaling can trigger hair growth and new hair follicle formation.

The K15 promoter effectively targets stem cells in the hair follicle bulge.

Inhibiting Bmp signaling disrupts hair growth and differentiation.

The research shows that a gene called Wnt3 affects hair growth and structure, causing short hair and balding when overactive.

Notch pathway activation causes abnormal hair layer development.