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IL-17 and certain immune cells are linked to more severe alopecia areata.

TRF1 is crucial for creating and maintaining stem cells and marks both pluripotent and adult stem cells.

Cancer cells often have more copies of TERT and TERC genes, which helps them grow and could affect patient outcomes.

Trodelvy™ helped some patients with advanced breast cancer, but had side effects.

The system allows precise control of gene expression in mouse skin, useful for studying skin biology.

Certain immune cells in atopic dermatitis skin could be targeted for treatment.

The combination therapy was effective and well-tolerated, especially in young patients.

Vaccines and targeting TrxR variants can help prevent cancer and reduce metastasis.

CTP-543 significantly improved eyebrow and eyelash regrowth and patient satisfaction in adults with alopecia areata.

The FDA approved the first gene therapy for a blood disorder after overcoming early challenges and demonstrating patient benefits.

A parasite-derived molecule speeds up skin healing and affects immune cell behavior without increasing scarring.

Lipid-antigen stimulation may play a role in folliculotropic mycosis fungoides.

Triptolide effectively and safely reduces actinic keratosis lesions in mice.

TR3 is mainly found in hair follicle stem cells and may be involved in hair loss.

Using polyethylenimine-DNA to deliver the hTERT gene can stimulate hair growth and may be useful in treating hair loss, but there could be potential cancer risks.

Sacituzumab tirumotecan shows promise in treating breast cancer with manageable side effects.

The Itpr3 gene causes a specific hair pattern in mice.

Mitochondrial features can predict colorectal cancer outcomes and improve immunotherapy.

Scientists turned rat thymus cells into stem cells that can help repair skin and hair.

A new genetic variant causes trichothiodystrophy in two brothers, but their mother may carry it without showing symptoms.

A parasite molecule can speed up skin healing and reduce scarring.

Tribuloside can increase skin pigmentation by enhancing melanin production and distribution.

Activating TLR3 boosts autophagy gene expression in skin cells.

rTMS may help treat trichotillomania in some patients.

TRIV-509 quickly improves skin barrier and cell health in atopic dermatitis.

Researchers created a mouse with the same mutation as humans with trichothiodystrophy, showing similar symptoms and confirming the condition is due to defects in DNA repair and gene activity.

TSC2-/meth cells can cause skin lesions, hair growth, and lung issues, and may be treated with chromatin remodeling agents.

Disrupting the Tsc2 gene in certain cells leads to thicker skin, larger hair, and changes in hair growth signaling, which can be partly reversed with specific treatment.

Trichothiodystrophy is a rare genetic disorder causing hair issues and sometimes linked to DNA repair defects.

The TGM3 gene's promoter region is key for skin and hair cell function and may aid gene therapy.