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Adult stem cells with Tp63 can form hair and skin cells when placed in new skin, showing they have hidden abilities for skin repair.

Trichoblastomas in rabbits are linked to uncontrolled embryonic hair growth and have distinct histological features.

Trichoscopy and trichogram are useful for diagnosing hair and scalp conditions.

Mouse zigzag hair bends form due to a 3-day cycle of changes in hair progenitors and their environment.

The hedgehog signaling pathway is crucial for hair growth but not for the initial creation of hair follicles.

High proliferation and cell delamination drive early skin development, while later stages may not rely on cell division orientation.

Growth hormone-releasing hormone (GHRH) boosts hair growth and human scalp hair follicles have their own growth hormone system.

Alopecia areata involves immune system issues and specific cell types that disrupt hair growth, leading to hair loss.

Blocking mTORC1 reduces skin tumor growth in mice.

BMP signaling is crucial for skin and hair growth.

Retinoic acid, glucocorticoids, and IGF1 increase IGFBP-3 production in human dermal papilla cells, affecting hair growth.

In alopecia areata, certain immune cells increase and express a protein linked to immune activation.

α-Phellandrene may help prevent hair loss by increasing growth factors and cell growth in hair cells through a specific signaling pathway.

CD8+ T cells drive alopecia areata, while regulatory T cells are protective.

Scientists found a new type of skin cell that could help with skin repair and these cells work better with a certain protein.

Trichostasis spinulosa mainly affects the nose, with many tiny hairs in one follicle, possibly influenced by hormones and sunlight.

Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

GLABRA2 represses root hair formation by inhibiting a specific gene.

CRH causes hair loss by reducing cell survival in hair follicles.

Overexpression of certain genes can shorten hair by disrupting the hair-growth cycle.

T cells affect skin cell genes in inflammatory diseases, and therapy can normalize these changes.

The TGM3 gene's promoter region is key for skin and hair cell function and may aid gene therapy.

Blocking the CXCL12–CXCR4 axis may help treat hair loss in alopecia.

Arrector pili muscle regulates hair follicle stem cells, DNA methylation needed for hair cycling, and Wnt/B-catenin signaling starts hair growth.

Feather patterns are influenced by enhancers and chromatin looping, and the structure of protein complexes important for hair growth has been detailed.

Overexpressing PKCα in mice skin increases inflammation but doesn't affect tumor growth.

Self-induced hair loss should be considered in patients with androgenetic alopecia.