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Mice without the p21 gene can fully regenerate injured ears due to reduced Sdf1 increase and leukocyte recruitment, suggesting new ways to induce tissue regeneration in mammals.

Putting thymidine dinucleotide on newborn mice's skin can delay and reduce skin cancer.

Krt15+ cells in the mouse intestine resist radiation and can start tumors.

Hair loss in certain young mice is linked to a specific gene and can be caused by lack of iron.

Mutations in β1 integrins cause embryonic death but have milder effects on skin.

Copper therapy improved health and enzyme activity in mice with copper deficiency.

The scraggly mutation causes hair loss and skin defects in mice.

Testosterone significantly affects urination differences between male and female mice.

CTHRC1 helps sweat glands recover by rebuilding nearby blood vessels.

Lack of vitamin D receptor causes hair loss in mice by allowing certain genes to overactivate.

Loss of the p53 gene alone causes tumors, and losing both p53 and Rb genes speeds up aggressive skin cancer.

Lack of 5α-Reductase type 1 can lead to insulin resistance and liver problems.

The flaky skin mouse mutation is a natural model for studying human psoriasis.

Loss of the p53 gene alone causes tumors, and losing both p53 and Rb genes speeds up aggressive skin cancer.

The p75 neurotrophin receptor is important for hair follicle regression by controlling cell death.

Early over-expression of FoxN1 harms immune and skin development.

Folliculin deficiency causes problems with cell division and positioning due to disrupted RhoA signaling and interaction with p0071.

RSPO1 mutations in certain patients lead to skin cells that don't develop properly and are more likely to become invasive, increasing the risk of skin cancer.

Krt15+ cells in mice can resist radiation, regenerate tissue, and start tumors, suggesting new cancer treatment targets.

Rac1 is vital for hair follicle health but not needed for skin maintenance.

The treatments stopped hair regrowth in mice.

The Oat mouse model shows mild retinal degeneration, useful for testing treatments.

Using testosterone-stimulated weanling rats can effectively replace castrated rats for anti-androgen testing, reducing animal stress.

A new mouse model for vitiligo helps study immune responses and potential treatments.

Certain genes are more active in baby scalp cells and can help grow hair when added to adult mouse skin cells.

Toll-like receptor 3 helps repair the skin barrier after UV damage.

Hairless mice are more vulnerable to Listeria infection, but gut microbiota can help reduce damage.

Identical twins had different symptoms because one had more cells with an extra chromosome fragment in different tissues.