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Thrombospondin-1 is crucial for hair follicle regression and reduced blood vessel growth during the catagen phase.

Certain gene variations in the Vitamin D Receptor are linked to higher risk of female hair loss.

Certain immune cells worsen post-surgery gut paralysis by activating a specific immune response.

Dormant melanoma cells in mice interact minimally with memory T cells due to a suppressive tumor environment.

CaBP1 and CaBP2 are important for continuous hearing by preventing inactivation of calcium currents in ear cells, with CaBP2 also able to restore hearing when reintroduced.

IL-18 signaling helps mature Tregs move into the thymus.

The vitamin D receptor helps control hair growth and may protect against certain skin tumors.

LPA3 is crucial for embryo implantation and links LPA to prostaglandin signaling.

Lack of Vitamin D receptor changes skin structure and increases certain immune cells in the skin.

α-parvin is necessary for skin and hair growth and for the correct orientation of skin cells.

TTD patients don't have a higher skin cancer risk because their main issue is with transcription, not DNA repair.

The study found that tight junctions reach the top layer of the skin's stratum granulosum, not just the second top layer as previously thought.

Activating TSPO helps protect the retina from damage caused by high eye pressure in glaucoma.

FERONIA and LRX proteins help control cell growth in plants by regulating vacuole expansion.

CaBP1 and 2 are necessary for maintaining calcium currents and hearing in inner ear cells.

RSPO1 mutations in certain patients lead to skin cells that don't develop properly and are more likely to become invasive, increasing the risk of skin cancer.

TRα and CRABPII genes change their activity levels during goat fetal skin development.

A unique gene mutation causes vitamin D-resistant rickets without causing hair loss.

Trapa bispinosa Roxb. extract may help reduce prostate size and cell growth in BPH.

KRT17 may be a new target for endometrial cancer treatment because it helps cancer cells move and form new blood vessels.

Vitamin D receptor can control the hairless gene linked to hair loss even without vitamin D.

People with acne have more CD4+ immune cells in their skin than healthy people.

Vitamin D boosts a specific gene activity in kidney cells that could improve heart and kidney function.

Different body areas have unique skin cell communication patterns, explaining why certain skin diseases occur in specific regions.

A vitamin D receptor mutation causes rickets and affects immune responses.

μ-opiate receptors in skin cells may affect skin health and healing.

Removing the VDR gene in skin cells reduces their growth and affects hair-related genes.