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Low Vitamin D receptor levels in breast cancer are linked to worse outcomes and more bone metastases, and could be a marker for prognosis.

Vitamin D receptor mutations can cause alopecia by affecting hair growth genes.

Vitamin D3 and its receptor help protect skin from UVB-related cancer.

Alopecia in these mice is caused by defective hair cycle communication due to missing vitamin D receptor function, not vitamin D levels.

Different Hairless isoforms affect Vitamin D receptor activity in hair regulation, with one repressing and the other stimulating it.

Vitamin D receptor helps prevent skin tumors.

The scant hair in snthr-1Bao mice is likely caused by a deletion affecting the Plcd1 gene.

A girl had rickets due to a gene mutation affecting vitamin D response.

The vitamin D receptor can regulate genes and affect hair and hormone levels without its usual activator.

Vitamin D Receptor is crucial for normal skin and hair growth.

Deleting vitamin D and calcium receptors in skin cells increases skin cancer risk by reducing DNA repair and stress response.

A specific gene mutation causes vitamin D resistance, but certain vitamin D analogs might help.

Vitamin D receptor is essential for preventing hair loss by regulating hair cycle stages.

Vitamin D Receptor is needed for hair growth in mice but not for skin stem cell maintenance.

Lack of Vitamin D receptor changes skin structure and increases certain immune cells in the skin.

Vitamin D receptor is essential for adult hair growth.

The vitamin D receptor is essential for skin stem cells to grow, move, and become different cell types needed for skin healing.

Vitamin D receptor helps prevent skin cancer and supports skin health.

Researchers created a mouse model of a type of rickets that does not cause hair loss.

Deleting part of a gene in mice causes wavy hair and high pup loss.

Vitamin D receptor is essential for healthy hair growth.

Vitamin D receptor may help protect against UV-induced skin cancer.

Mutations in the Vitamin D receptor gene can cause hair loss similar to mutations in the Hairless gene.

The vitamin D receptor is essential for normal hair growth, even without its usual binding.

The vitamin D receptor helps control hair growth and may protect against certain skin tumors.

A protein called PCBP2 controls the production of a hair growth protein by interacting with its genetic message and is linked to hair loss when this control is disrupted.

Vitamin D receptor and mediator 1 are crucial for healthy skin and hair growth.

Vitamin D receptor gene changes don't affect alopecia areata risk.

Disrupting Acvr1b in mice causes severe hair loss and thicker skin.

A gene mutation causes early keratinocyte maturation leading to hair loss in Olmsted syndrome.