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The TRPV3 channel structure changes linked to severe itch and hyperkeratosis were identified using cryo-EM.

A unique gene mutation was found in a family with monilethrix.

Human Schwann cells can be quickly made from hair follicle stem cells for nerve repair.

New methods for skin and nerve regeneration can improve healing and feeling after burns.

Hair follicle stem cells can help repair nerve and spinal cord injuries.

A 7-year-old girl was diagnosed with Netherton's Syndrome, shown by skin and hair symptoms.

TRPV3 channels are involved in skin processes and are affected by shear stress, influencing itch and mechanotransduction.

Neurotoxins can affect neurotransmitter release and have potential in treating muscle, pain, and cancer conditions, but more research is needed on how they work.

Tenascin is present in normal skin and certain skin tumors but not in sebaceous glands or their tumors.

Trichohyalin is modified by enzymes to form strong structures in hair cells.

Higher CSF 5-HIAA levels may predict better response to serotonin re-uptake inhibitors in trichotillomania.

Meis2 is essential for touch sensation and proper nerve connection to touch receptors in certain skin areas of mice.

Proper diet management is crucial for preventing severe symptoms in phenylketonuria.

Skin pigmentation increases in CRPS-affected areas and improves with symptom relief.

Skin lesions in Carney Complex are caused by a gene change in some skin cells that leads to increased pigmentation and may lead to tumors.

Alopecia areata patients experience more psychological distress, linked to certain neurotrophin levels.

Beta-trace protein may help diagnose and predict treatment response in certain nerve disorders.

Lysophosphatidic acid affects sensory neurons and may cause neuropathic pain and itch.

The nervous system helps control stem cell behavior and immune responses, affecting tissue repair and maintenance.

The sympathetic nervous system is linked to scalp hair loss.

The ID2 gene can help distinguish between sensitizers and irritants in skin cells.

Keratin gel may help repair damaged nerves.

Pathologic tissues have more soluble proteins than normal tissues.

Mutations in the hHb6 gene cause the hair disorder monilethrix.

Neuronatin is found in specific cells within rat testis, hair follicles, tongue, and pancreas, suggesting it has various roles in tissue development and function.

High doses of tryptophan may cause eosinophilic fasciitis.

Melanocyte activity in hair follicles is linked to the hair growth cycle, being active in growth phases and inactive in rest phases.

A new mutation, Glu402Lys, in hair keratin is linked to variable symptoms of monilethrix.

A new enzyme in rats may help regulate hair growth.

Light can turn on hair growth cells through a nerve path starting in the eyes.