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Faulty Notch signalling may cause hair follicle changes and inflammation in hidradenitis suppurativa.

The protein DDX6 helps keep skin cells renewing properly by controlling the production of certain other proteins and breaking down those that cause cells to mature too quickly.

Cells in hair die by breaking down their DNA and mitochondria as they form keratin.

Quiescent cells have increased mitochondrial activity and ECM gene expression, but reduced glycolysis.

Blocking autophagy increases survival of inner ear hair cells exposed to gentamicin.

Inhibiting the PI3K/Akt pathway may help prevent radiation-induced liver injury.

Overexpressing SSAT enzyme reduces prostate tumor growth in mice.

Defective nuclear transport may cause gene expression changes in Progeria.

848 genes related to fat and metabolism are less active in people with Central Centrifugal Cicatricial Alopecia.

The mouse model showed defects in adult stem cell maintenance related to Hutchinson-Gilford progeria syndrome.

The study found that skin inflammation and immune response pathways are activated in Solar Urticaria patients after UV exposure, unlike in healthy people.

Type II keratins are uniquely phosphorylated during stress and mitosis, affecting their structure and function.

Antizyme slows skin tumor growth by reducing cell growth in mice.

Folliculin helps regulate energy and nutrient sensing, impacting Birt–Hogg–Dubé syndrome.

Cadd4 effectively reduces cholesterol levels without side effects.

LGR5 mainly stays inside cells, moving to the trans-Golgi network, and this process is important for its role in cell signaling.

UVB exposure increases skin proteins for retinoic acid synthesis and shifts their location, possibly affecting skin repair.

Alopecia areata patients show unique protein activity patterns, suggesting imbalanced signaling pathways.

Targeting the IL-23/IL-17 pathway effectively treats several inflammatory skin diseases.

A new mutation in the PLEC gene causes a rare condition with skin blistering, muscle weakness, and hair loss.

Stress in hair follicle cells increases certain immune-related proteins, which might contribute to hair loss conditions.

The protein's size was reduced, but more work is needed to confirm its function.

Removing the ROBO4 gene in mice reduces skin inflammation and hair loss by affecting certain inflammation pathways and gene expression.

The research showed that CRISPR/Cas9 can fix mutations causing a skin disease in stem cells, which then improved skin grafts in mice, but more work on safety and efficiency is needed.

Baricitinib reduces inflammation and improves cell health in premature aging cells.

UTX is crucial for skin differentiation and health, especially in females.

IGFBP5 may be a potential target for Parkinson's treatment by reducing neuron death.

Treatments for burn wounds in rats should be timed according to whether autophagy or apoptosis is more active at certain times after the burn.

Deleting Twist1 in skin cells reduces UVB-induced skin cancer risk.