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Autophagy, a cell process, helps activate hair growth stem cells and promote hair growth by controlling glycolysis, a type of cell metabolism.

Overexpressing IGF-1 in mice leads to skin abnormalities and tumors.

Exosomal miR-199a-3p from dermal papilla cells helps control hair color by affecting melanocytes.

Arabidopsis plants adjust gene expression to maintain balance when auxin pathways are disrupted.

Cathepsin L deficiency causes large, abnormal cell structures and health issues in mice.

KAP8.1 protein is crucial for hair structure and interacts with keratin 85.

Mutations in the hHb6 gene cause the hair disorder monilethrix.

EGFR helps hair follicles transition properly by controlling Stathmin levels.

Epristeride's metabolism in zebrafish helps improve doping detection methods.

Igf1r helps regulate hair growth cycles.

PLAU and SerpinB2 affect cell death differently in various forms of leprosy and could be targets for new treatments.

ETS2 is crucial in squamous cell carcinoma development and could be a therapeutic target.

A new method creates unique chemical structures that could improve drug discovery.

The mK6irs1/Krt2-6g gene likely causes wavy hair in mice.

A new method efficiently creates biaryl N-oxides with potential for cancer treatment and drug development.

Rapamycin treatment helps reduce brain inflammation and symptoms of mitochondrial disease by blocking specific pathways in mice.

Injecting specific oligonucleotides can change hair growth and structure by altering a gene.

The protein SLC1A3 is important for activating skin stem cells and is necessary for normal hair and skin growth in mice.

Epidermal Wnt/β-catenin signaling controls fat cell formation and hair growth.

Involucrin is crucial for skin cell maturation and protection.

Poly(ADP-ribose) synthesis is linked to skin cell differentiation.

The 736T>A mutation in the LIPH gene is common in Japanese people with autosomal recessive woolly hair.

INTASYL is a promising, adaptable RNAi technology for treating skin cancers.

The NUDT15 R139C variant causes thiopurine-induced leukocytopenia through a different mechanism than previously thought in Japanese patients with inflammatory bowel disease.

Blocking mTORC1 reduces skin tumor growth in mice.

SkQ1 antioxidant improved health and lifespan in mice.