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The human scalp hair bulb contains different types of melanocytes with varying abilities to produce melanin.

Tgm2 helps stabilize dying cells and aids fibroblast attachment to the extracellular matrix.

NKG2D+CD4+ T cells are higher in alopecia areata patients and may be involved in the disease.

Understanding T cells and signaling pathways can lead to better treatments for hair loss.

A new type of nerve cell involved in itch perception was discovered.

Researchers developed a 3D skin model with its own immune and blood vessel cells to better understand skin health and disease.

Using regulatory T cells and Rapamycin together improves chronic graft-versus-host disease treatment outcomes in mice.

γδTregs can protect hair follicles from alopecia areata and may help regrow hair.

A mouse model for studying scleroderma in chronic graft-versus-host disease was successfully created.

Alopecia areata involves immune system changes, especially in severe cases, with potential new treatment targets identified.

Amotosalen-treated donor T-cells can prevent late CMV infection after bone marrow transplants.

CD34+ and CD34- melanocyte stem cells have different regenerative abilities.

Adult stem cells with Tp63 can form hair and skin cells when placed in new skin, showing they have hidden abilities for skin repair.

CD4 T cells need IFN-γ to cause hair loss in alopecia areata.

Trichoblastic carcinoma may be a distinct type of skin cancer different from basal cell carcinoma.

BST2 is highly expressed in certain immune cells in alopecia areata, suggesting a role in the disease.

The antibody created from BCC tissues reacts similarly to both BCC and hair follicles, suggesting BCC may come from hair follicle cells.

Increased Toll-like receptors in blood cells may contribute to alopecia areata and could be a target for new treatments.

AP-1 and TGFß work together to drive resistance in basal cell carcinoma, suggesting new treatment options.

The muscle damage was caused by T-cell large granular lymphocytic leukemia, not dermatomyositis.

IL-1 signaling is crucial for hair follicle stem cell growth and wound healing.

CD80CD86 deficiency causes hair loss by disrupting regulatory T cells.

The type of tumor suppressor gene lost affects the behavior of skin cancer.

Vesiculobullous lesions should be considered part of canine cutaneous epitheliotropic T-cell lymphoma.

Standard dosing is more effective for severe alopecia areata in children, but unorthodox dosing can be safe and considered if needed.

Newly designed proteins can effectively degrade specific proteins in cells, offering a promising alternative for targeted protein degradation.

A specific group of skin stem cells was found to help maintain hair follicle cells.

Certain microRNAs found in normal cells can effectively suppress various cancers.

The human scalp has different types of pigment cells in hair follicles with varying abilities to produce pigment.