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A Chinese family had a child with a specific gene mutation causing vitamin D-resistant rickets, but the child improved with calcium and low-dose calcitriol.

The study found that certain mutations in the vitamin D receptor can cause rickets and potentially affect hair growth.

Certain gene combinations may affect prostate cancer risk, and GSTP1 methylation is linked to tumor aggressiveness.

New vitamin D3 forms need the vitamin D receptor to reduce fibrosis in human cells.

Low Vitamin D receptor levels in hair follicles may contribute to alopecia areata.

Reduced Vitamin D receptor expression may cause hair loss through the Wnt/b-catenin pathway.

p63 and p73 regulate the vitamin D receptor, affecting cancer cell behavior and vitamin D sensitivity.

Certain VDR gene changes can affect melanoma risk.

Nocturnal calcium infusions improved a boy's severe rickets without causing hair loss.

The vitamin D receptor is involved in multiple body functions beyond calcium regulation, including immune response and rapid reactions not related to gene activity.

The vitamin D receptor is essential for normal hair growth, even without its usual binding.

Vitamin D receptor helps control hair growth and could be used to treat certain skin tumors.

Expressing the human vitamin D receptor in skin cells prevents hair loss in certain mice.

Alopecia in VDR knockout mice is due to a defect in hair cycle initiation, not keratinocyte issues.

Vitamin D receptors in hair follicles change with the hair cycle, affecting hair growth.

A specific gene mutation causes vitamin D resistance, but certain vitamin D analogs might help.

Vitamin D receptor binds similarly to natural and synthetic ligands, affecting gene regulation.

Targeting vitamin D and androgen receptors may effectively treat triple-negative breast cancer, especially with chemotherapy.

Vitamin D receptor is essential for hair growth signaling.

Vitamin D and its receptor may help prevent skin cancer.

Vitamin D receptor is essential for hair growth and preventing hair loss.

A new gene mutation causes vitamin D resistance and rickets, treatable with calcium therapy.

A mutation in the vitamin D receptor causes severe resistance to vitamin D, affecting bone health but not hair growth.

A specific gene mutation causes vitamin D-resistant rickets and hair loss.

Most Hairless gene mutations reduce its ability to work with the Vitamin D Receptor, which might explain a certain type of hair loss.

A girl had rickets due to a gene mutation affecting vitamin D response.

A new gene mutation causes vitamin D resistance and hair loss in two unrelated girls.

Vitamin D receptor is crucial for healthy hair growth and preventing hair loss.

Alopecia in VDR knockout mice is due to impaired hair cycle initiation, not keratinocyte issues.

Vitamin D Receptor is needed for hair growth in mice but not for skin stem cell maintenance.