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Alpha-difluoromethylornithine prevents cancer in mice but causes hair loss.

Langerhans' cells are not responsible for depigmentation in this mouse model.

The Lanceolate hair-J mutation in mice mimics human hair disorders like Netherton's syndrome.

The Kras mutation changes normal cell signals, leading to disrupted tissue structure and potential cancer.

Prolactin affects when mice shed and grow hair.

Hairless HRS/J mice resist Bacillus anthracis skin infections due to high numbers of immune cells, not because they lack hair follicles.

Hair follicles in the back of the rosette fancy mouse have reversed orientations due to a gene mutation.

Alopecia areata is an autoimmune disease that targets hair follicles.

Mice without the enzyme HSD17B3 still produce normal testosterone, suggesting they have different ways to make it compared to humans.

Vaccines and targeting TrxR variants can help prevent cancer and reduce metastasis.

CD44 variant changes start alopecia areata, but don't maintain it.

Blocking mTORC1 reduces skin tumor growth in mice.

Smad2 and Smad3 are essential for normal skin development, and their absence causes severe skin issues and cancer.

The study concludes that a genetic mutation in TFM mice leads to reduced androgen receptor activity, affecting the body's response to male hormones.

Zebrafish help understand genetic causes of skin pigment disorders like albinism.

Signals from skin cells controlled by Rac proteins help turn certain precursor cells into white fat cells.

The 4C32 gene may help in mouse skin development and differentiation.

Mice can correct hair follicle orientation without certain genes, but proper overall alignment needs those genes.

Mice with LSS deficiency showed hair loss and cataracts, similar to humans, and can help in understanding and treating this condition.

Harlequin mutant mice have hair loss due to low AIF protein levels and retroviral element activity.

HSD11b1 affects skin nerves and increases non-histaminergic itch.

MED1 affects skin wound healing differently in young and old mice.

Gene expression, especially Dkk4, is key to cat color patterns.

FTase and GGTase-I are essential for skin keratinocyte health.

Loss of a specific protein in skin cells causes symptoms similar to psoriasis.

Scientists made a mouse that shows how a specific protein in the skin changes and affects hair growth and shape.

Male-to-female skin grafts in mice are rejected due to sex-linked antigens.

A new genetic mutation in the ODC1 gene causes developmental delay and other symptoms in a young girl.

The animal models successfully simulated dry eye related to sex steroid deficiency.

NSG mice had the most mites, and genetic factors affect immune response and susceptibility.