Search

everythinglearnresearchcommunity

for

Search:↓

Low Vitamin D receptor levels in breast cancer are linked to worse outcomes and more bone metastases, and could be a marker for prognosis.

Hairless protein helps control hair growth by regulating vitamin D receptor activity.

The mutation causes hairless mice due to mislocalized and dysfunctional HR protein.

Mutations in the hairless protein gene cause hair loss.

A unique gene mutation causes vitamin D-resistant rickets without causing hair loss.

Stress activates a special function of the Vitamin D receptor with the help of c-Jun, which can also prevent cell death.

High CRP levels could indicate vitamin D deficiency in people with alopecia areata.

The mutant HR bmh protein mis-localizes in cells, affecting skin and hair development.

Vitamin D receptor and hairless protein are essential for hair growth.

Vitamin D receptor mutations can cause alopecia by affecting hair growth genes.

Most Hairless gene mutations reduce its ability to work with the Vitamin D Receptor, which might explain a certain type of hair loss.

A genetic mutation caused severe rickets and alopecia in an Indian patient, but high-dose calcium and phosphate treatment improved their condition.

Hairless protein can block vitamin D activation in skin cells.

Certain mutations in the hairless protein disrupt its ability to regulate the hair cycle.

The HR protein's role as a repressor is essential for controlling hair growth.

Hairless protein is crucial for healthy skin and hair, and its malfunction can cause hair loss.

Two siblings with a genetic mutation had a form of rickets that doesn't respond to vitamin D.

Vitamin D receptor is essential for proper skin healing after injury.

The mutant HR bmh protein affects hair follicle formation by failing to repress vitamin D receptor activity.

ΔNp63α helps control a protein that stops cancer cells from spreading.

Hairless protein can both repress and activate vitamin D receptor functions, affecting gene regulation.

Vitamin D affects Msx1 protein expression and may influence mineralized tissue health.

Vitamin D helps skin stem cells heal wounds by working with a key skin protein.

Low Vitamin D receptor levels in hair follicles may contribute to alopecia areata.

The R343H mutation in the VDR gene causes vitamin D-resistant rickets with alopecia by impairing specific gene activity.

Vitamin D3 and its receptor help protect skin from UVB-related cancer.

A mutation in the VDR gene affects hair cycling without needing ligand binding.

p63 and p73 regulate the vitamin D receptor, affecting cancer cell behavior and vitamin D sensitivity.

Removing part of the vitamin D receptor stops vitamin D from working properly.

A mutation in the vitamin D receptor causes severe resistance to vitamin D, affecting bone health but not hair growth.