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The vitamin D receptor is essential for skin stem cells to grow, move, and become different cell types needed for skin healing.

Vitamin D and calcium are essential for effective wound healing and hair growth.

Vitamin D and its receptor are crucial for skin and hair health.

Vitamin D3 and its receptor help protect skin from UVB-related cancer.

Vitamin D Receptor is crucial for hair follicle shrinkage and cell death, affecting hair growth.

Removing vitamin D and calcium receptors in mice skin cells slows down skin wound healing.

Removing part of the vitamin D receptor stops vitamin D from working properly.

Vitamin D and calcium are crucial for proper skin wound healing.

Stress activates a special function of the Vitamin D receptor with the help of c-Jun, which can also prevent cell death.

The mTOR signaling pathway is crucial for hair health and targeting it may lead to new hair loss treatments.

New vitamin D3 forms need the vitamin D receptor to reduce fibrosis in human cells.

Blocking Oncostatin M's role in the JAK-STAT pathway can stimulate hair growth in mice.

Double-stranded RNA activates a pathway that causes a skin protein to be expressed in the wrong place.

Blocking Prostaglandin D₂ (PGD₂) could help treat hair loss.

A Chinese family had a child with a specific gene mutation causing vitamin D-resistant rickets, but the child improved with calcium and low-dose calcitriol.

Wnt signaling is crucial for skin development and health, and its disruption can cause skin diseases.

The HR protein helps hair grow by blocking a hair growth inhibitor, aiding in hair follicle regeneration.

ΔNp63α helps control a protein that stops cancer cells from spreading.

Senescent melanocytes can boost hair growth by activating hair stem cells.

Hair loss involves immune responses, inflammation, and disrupted signaling pathways.

1α,25-dihydroxyvitamin D3 directly affects cartilage growth and development.

The R343H mutation in the VDR gene causes vitamin D-resistant rickets with alopecia by impairing specific gene activity.

Hair loss in Androgenetic alopecia (AGA) is due to altered cell sensitivity to hormones, not increased hormone levels. Hair growth periods shorten over time, causing hair to become thinner and shorter. This is linked to miscommunication between cell pathways in hair follicles. There's also a change in gene expression related to blood vessels and cell growth in balding hair follicles. The exact molecular causes of AGA are still unclear.

Targeting specific cell interactions may help treat skin fibrosis.

Certain VDR gene changes can affect melanoma risk.

Activin A is important for creating new hair follicles.

The vitamin D receptor helps maintain hair and bone health even without binding vitamin D.

Skin cells produce and activate vitamin D, which regulates skin functions and supports hair growth.

Vitamin D has potential benefits for cancer prevention, heart health, diabetes, obesity, muscle function, skin health, and immune function, but clinical results are mixed and more research is needed.

Nuclear hormone receptors play a significant role in skin wound healing and could lead to better treatment methods.