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Estrogen receptor α controls hair growth cycles and skin thickness in male mice.

Lgr5 and the vitamin D receptor are key in controlling skin inflammation and tumor risk in mice.

Brindled mice show abnormal catecholamine neuron development due to copper deficiency.

Progesterone's seizure-reducing effects are mainly due to allopregnanolone, not progesterone receptors.

NKIRAS2 can suppress certain skin tumors but its effect on cancer varies with context and expression level.

Researchers fixed gene mutations causing a skin disease in stem cells, which then improved skin grafts in mice.

Signals from skin cells controlled by Rac proteins help turn certain precursor cells into white fat cells.

Hairless mice lack fur due to a genetic mutation affecting skin response, not hormone issues.

External factors can cause skin cancer cells that usually don't spread to grow and form tumors in mice.

Tranexamic acid turns white hair brown in certain mice by affecting specific proteins.

Progeroid mouse models show signs of early aging similar to humans, helping us understand aging better.

Foxn1 is essential for hair pigmentation by directing pigment transfer to hair cells.

Lethal-milk mice produce zinc-deficient milk, causing health issues in pups unless supplemented with zinc.

Mice with CBS deficiency are healthier on a low-methionine diet.

The research showed that CRISPR/Cas9 can fix mutations causing a skin disease in stem cells, which then improved skin grafts in mice, but more work on safety and efficiency is needed.

EGFR helps mouse hair follicles stop growing by reducing certain growth regulators.

SKH1 hairless mice have identifiable epidermal stem cells with specific markers.

Injecting specific oligonucleotides can change hair growth and structure by altering a gene.

The Foxn1 gene is essential for normal nail and hair development.

The PML protein helps prevent skin cancer in mice.

Blocking a specific protein signal can make hair grow on mouse nipples.

Meis2 is essential for touch sensation and nerve function in mice.

Researchers successfully used nude mice to study human hair growth, which could help with future hair research.

CRISPR/Cas9 and prime editing can potentially fix skin disorder genes safely and effectively.

The balance between cell renewal and differentiation controls the growth of cancerous cells in mouse skin.

Scientists made a mouse model of a serious skin cancer by changing skin cells with a virus and a specific gene, which is similar to the disease in humans.

Adult spiny mice recover better from heart attacks than common lab mice.

Antiviral drugs, especially daclatasvir, may be a new treatment for a rare skin disease, improving survival and reducing symptoms in mice.

Removing MCPIP1 from myeloid cells in mice leads to hair loss and prevents skin tumors but causes pigmented spots.