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Certain gene combinations may affect prostate cancer risk, and GSTP1 methylation is linked to tumor aggressiveness.

Low Vitamin D receptor levels in hair follicles may contribute to alopecia areata.

Reduced Vitamin D receptor expression may cause hair loss through the Wnt/b-catenin pathway.

Nocturnal calcium infusions improved a boy's severe rickets without causing hair loss.

Vitamin D receptor knockout mice have significant motor impairments but no cognitive deficits.

The vitamin D receptor is essential for normal hair growth, even without its usual binding.

Vitamin D and its receptor are crucial for bone health and can be influenced by certain fats and curcumin.

Alopecia in these mice is caused by defective hair cycle communication due to missing vitamin D receptor function, not vitamin D levels.

Diet can prevent bone issues but not hair loss in mice lacking vitamin D receptors.

Expressing the human vitamin D receptor in skin cells prevents hair loss in certain mice.

Alopecia in VDR knockout mice is due to a defect in hair cycle initiation, not keratinocyte issues.

Vitamin D receptor is essential for healthy bones and skin.

Vitamin D receptor mutations can cause alopecia by affecting hair growth genes.

Vitamin D receptors in hair follicles change with the hair cycle, affecting hair growth.

A specific gene mutation causes vitamin D resistance, but certain vitamin D analogs might help.

VDDR I and II are genetic disorders affecting vitamin D use, causing rickets, with VDDR I treatable by vitamin D supplements and VDDR II needing high doses and calcium.

Vitamin D receptor is essential for hair growth signaling.

Vitamin D receptor is essential for proper skin healing after injury.

Vitamin D receptor is essential for hair growth and preventing hair loss.

Vitamin D receptor is essential for healthy hair growth.

A new gene mutation causes vitamin D resistance and rickets, treatable with calcium therapy.

Vitamin D receptor helps control skin cell growth and hair development.

A specific gene mutation causes vitamin D-resistant rickets and hair loss.

The vitamin D receptor can regulate genes and affect hair and hormone levels without its usual activator.

A girl had rickets due to a gene mutation affecting vitamin D response.

A new gene mutation causes vitamin D resistance and hair loss in two unrelated girls.

Vitamin D receptor is crucial for healthy hair growth and preventing hair loss.

Alopecia in VDR knockout mice is due to impaired hair cycle initiation, not keratinocyte issues.

Hairless protein can both repress and activate vitamin D receptor functions, affecting gene regulation.

Four patients with a type of rickets and hair loss had different mutations in their vitamin D receptor gene, causing it to not work properly.