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The mutation causing Hutchinson-Gilford progeria syndrome leads to severe skin problems and early death in mice.

Mice lacking a key DNA methylation enzyme in skin cells have a lower chance of activating stem cells necessary for hair growth, leading to progressive hair loss.

Epidermis and dermis cells together can regenerate hair follicles.

DNA virus-induced tumors have consistent isozyme profiles, unlike other tumor types.

The Foxn1 gene is essential for normal nail and hair development.

A gene mutation causes a rare hereditary hair loss, offering potential for new treatments.

Certain mutations in a hair growth-related gene cause a type of genetic hair loss.

Deleting Rac1 in the skin depletes stem cells and damages hair follicles.

The gene Prss53 affects hair shape and bone development in rabbits.

Deleting Smad4 and PTEN genes in mice causes rapid, invasive stomach cancer.

Radiation significantly slows down wound healing in mice.

A new pair of mouse keratins, 65 kD and 48 kD, are found in specific skin areas and are linked to a unique skin differentiation type.

Dermal lymphatic vessels help hair growth by affecting hair cycle phases.

Blocking CRF-R1 can reduce alcohol intake in stressed mice.

Basal cell carcinomas in mice can start from hair follicle stem cells and other skin cell types, depending on signaling levels.

The hepatitis B vaccine did not cause hair loss in the tested mice.

Msx-2 gene removal speeds up skin wound healing in mice.

Dopaminergic neurons in the gut have distinct subtypes, some releasing both dopamine and acetylcholine.

Androgen increases VEGF expression but not VEGFR-2 in rat corpus cavernous.

The method successfully isolates hair follicle stem cells and skin cells from mice for research.

Removing a specific gene in certain skin cells causes hair loss on the body by disrupting normal hair development.

Key genes linked to hair growth and cancer were identified in hairless mice.

A Gsdma3 mutation causes hair loss due to stem cell damage from skin inflammation.

Signals from skin cells controlled by Rac proteins help turn certain precursor cells into white fat cells.

Involucrin is a useful marker for keratinocyte differentiation in mice.

Loss of Dnmt3a and Dnmt3b increases aggressive skin tumors by affecting PPAR-γ.

Transferrin receptor 1 is essential for normal function in the intestines, pancreas, and skin.

IL-1 receptor absence in mice leads to skin cysts and changes in immune response after UVB exposure.

The HCR gene contributes to psoriasis risk.