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The vitamin D receptor helps control hair growth and may protect against certain skin tumors.

Azelaic acid and vitamin B6 together may improve hair growth.

A balanced diet with essential vitamins, especially B-complex, prevents hair loss.

Low vitamin D levels may contribute to female pattern hair loss.

Expressing the human vitamin D receptor in skin cells prevents hair loss in certain mice.

Vitamin D and calcium are essential for effective wound healing and hair growth.

Measuring 24-OHase induction helps identify defects in vitamin D processing and predict treatment response.

Vitamin B12 deficiency can cause skin and hair color changes, which are reversible with treatment.

Vitamin D receptor is essential for hair growth signaling.

Topical vitamin D3 does not prevent hair loss from chemotherapy.

Most Hairless gene mutations reduce its ability to work with the Vitamin D Receptor, which might explain a certain type of hair loss.

A girl had rickets due to a gene mutation affecting vitamin D response.

Synthetic hormone treatment reduces melanin and hair growth in rats.

Four patients with a type of rickets and hair loss had different mutations in their vitamin D receptor gene, causing it to not work properly.

Certain mutations in the hairless protein disrupt its ability to regulate the hair cycle.

Excess vitamin A changes skin and hair follicle development and affects polysaccharide distribution.

Different Hairless isoforms affect Vitamin D receptor activity in hair regulation, with one repressing and the other stimulating it.

High Nesfatin-1 and low Vitamin D may increase blood pressure and heart rate in women with PCOS.

Vitamin B12 deficiency can cause skin darkening and increased blood vessels, but treatment can improve symptoms.

Vitamin B12 deficiency can cause skin darkening and white hair, but it's reversible.

Lack of Vitamin D receptor changes skin structure and increases certain immune cells in the skin.

Vitamin D and calcium are important for quick and effective skin wound healing.

Alopecia areata is not linked to vitamin B12 deficiency.

Early detection of specific VDR mutations is crucial for effective treatment and better dental outcomes in children with hereditary vitamin D–resistant rickets.

A vitamin D receptor mutation causes rickets and affects immune responses.

Vitamin D3-activated cell byproduct promotes hair growth in mice by increasing blood vessel growth.

Vitamin B12 deficiency can cause skin and hair color changes, which can be reversed with treatment.

Vitamin D receptor can control the hairless gene linked to hair loss even without vitamin D.

Low Vitamin D receptor levels in breast cancer are linked to worse outcomes and more bone metastases, and could be a marker for prognosis.

Child with rickets improved with a specific vitamin D therapy, but alopecia did not change.