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Vitamin D3 and parathyroid hormone-related peptide may have important uses in skin treatments.

A girl with rickets and hair loss was found to have a rare condition where her body didn't respond to vitamin D treatment.

Vitamin D boosts a specific gene activity in kidney cells that could improve heart and kidney function.

Hair loss can be a key sign of a rare type of rickets when vitamin D treatment doesn't work and advanced tests aren't available.

The enzyme 25 Hydroxyvitamin D 1 α-Hydroxylase is essential for healthy skin and recovery after skin damage.

The vitamin D receptor is involved in multiple body functions beyond calcium regulation, including immune response and rapid reactions not related to gene activity.

The R343H mutation in the VDR gene causes vitamin D-resistant rickets with alopecia by impairing specific gene activity.

The vitamin D receptor has many roles in the body beyond managing calcium, affecting the immune system, hair growth, muscles, fat, bone marrow, and cancer cells.

Mice without the vitamin D receptor have bone issues and other health problems, suggesting vitamin D is important for preventing various diseases in humans.

Skin cells produce and activate vitamin D, which regulates skin functions and supports hair growth.

Vitamin D and its receptor regulate skin functions like cell growth, immunity, hair cycle, and tumor prevention.

A child with rickets and hair loss might have a rare type of rickets that doesn't improve much with usual vitamin D and calcium treatment.

A child with a rare type of rickets showed some improvement with high doses of vitamin D, but such conditions often respond poorly to treatment.

The vitamin D receptor can affect gene activity even without its usual hormone in hair and skin.

Vitamin D receptor helps prevent skin cancer and supports skin health.

The vitamin D receptor can act without its usual activating molecule, affecting hair growth and skin cancer, but its full range of actions is not well understood.

Vitamin D receptor is crucial for skin wound healing.

Researchers created a mouse model of a type of rickets that does not cause hair loss.

OCT binds strongly to hair sheath cells and may affect skin and hair growth with fewer side effects than vitamin D3.

A boy with severe Vitamin D-resistant rickets did not respond to treatment and lacked a common symptom, suggesting a need for alternative treatments.

Vitamin D compounds may help treat psoriasis by promoting skin cell differentiation.

Redheaded people may have evolved to efficiently make vitamin D in areas with less sunlight.

1α,25-dihydroxyvitamin D3 directly affects cartilage growth and development.

Oral Vitamin D can improve hair density and reduce hair loss in Telogen Effluvium patients.

Men and premenopausal women in Korea show different patterns in iron and vitamin D levels, with no clear pattern for postmenopausal women.

Belimumab effectively controls SLE disease activity and reduces steroid use.

A new gene mutation causes vitamin D resistance and rickets, treatable with calcium therapy.

A unique gene mutation causes vitamin D-resistant rickets without causing hair loss.

New mutations in the VDR gene cause vitamin D-resistant rickets without hair loss.

Vitamin D receptor interacts with certain dietary components to help prevent diseases and regulate hair growth.