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A new gene mutation causes vitamin D resistance and hair loss in two unrelated girls.

Alopecia indicates more severe resistance to 1,25-dihydroxyvitamin D.

Patients with this syndrome can have different responses and worsening resistance to treatment over time.

Vitamin D receptor interacts with certain dietary components to help prevent diseases and regulate hair growth.

Vitamin D and calcium are essential for normal hair growth.

Vitamin D3 may prevent hair loss from chemotherapy, but side effects and cancer cell protection are concerns.

Vitamin D3 speeds up skin healing by helping skin stem cells grow and develop.

A Pomeranian dog had rickets due to a new gene mutation, leading to severe symptoms and euthanasia.

Vitamin D3 and its receptor help protect skin from UVB-related cancer.

A mutation in the vitamin D receptor causes severe resistance to vitamin D, affecting bone health but not hair growth.

VDDR I and II are genetic disorders affecting vitamin D use, causing rickets, with VDDR I treatable by vitamin D supplements and VDDR II needing high doses and calcium.

Topical calcitriol-analogs can reduce hair loss caused by chemotherapy.

Vitamin D receptor helps control skin cell growth and hair development.

RXR agonists may promote hair growth in humans.

Vitamin D-dependent rickets Type II causes bone problems and hair loss, and doesn't improve with Vitamin D treatment.

Vitamin D receptor can control the hairless gene linked to hair loss even without vitamin D.

Three siblings with a genetic form of rickets showed different symptoms of the disease.

A 2-year-old girl with a rare vitamin D disorder had rickets and hair loss, but treatment was ineffective due to poor compliance.

Vitamin D may help protect against nonmelanoma skin cancer.

Vitamin D3 analogs can stimulate hair growth in mice with alopecia.

Vitamin D is important for skin cell growth and health, and its active form and receptor play key roles in skin and hair processes.

A child with rickets and hair loss might have a rare type of rickets that doesn't improve much with usual vitamin D and calcium treatment.

Vitamin D supplements should be used cautiously in sarcoidosis patients, with regular monitoring to avoid high calcium levels.

Patients with the same vitamin D receptor mutation showed different symptoms due to other factors.

Hairless protein can both repress and activate vitamin D receptor functions, affecting gene regulation.

Vitamin D might help with hair growth and hair loss treatments.

Hair follicle cells can become bone-like cells, useful for bone repair.

Vitamin D receptor may help protect against UV-induced skin cancer.

Vitamin D3 ointment improved skin bumps on the chin but didn't give lasting results after stopping use.

Four patients with a type of rickets and hair loss had different mutations in their vitamin D receptor gene, causing it to not work properly.