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Glioblastoma cells can make androgens, which might help the tumor grow.

A more sensitive assay was developed to detect enzyme activity converting arginine to citrulline in hair follicles.

A specific gene mutation in KRT86 is linked to hair disorder in a Chinese Han family.

The method effectively identifies banned substances in hair loss and skin disease cosmetics.

USPlus PRO saw palmetto extract improves urinary symptoms and is safe for men.

People with vitiligo or alopecia areata have higher anxiety and depression than healthy people.

Men with male pattern baldness had higher body fat and obesity-related measurements but similar heart fat and artery thickness compared to healthy men.

TPA strongly increases ODC activity in certain skin cells, potentially aiding tumor growth.

Ornithine decarboxylase is crucial for hair growth and follicle development.

An over-the-counter vitamin/mineral supplement improved hair loss and blood sugar control in a woman with non-classic 11-hydroxylase deficiency.

Increased HSD11B1 enzyme expression is linked to higher body fat and insulin resistance.

Idiopathic hirsutism may be linked to increased enzyme activity.

Three new gene mutations cause rickets and hair loss, treatable with high calcium and calcidol, but hair regrowth is rare.

Minoxidil reduces lysine hydroxylase in skin cells.

Human sebaceous glands contain enzymes that affect androgen production and may influence sebum production and acne.

The enzymes Tet1, Tet2, and Tet3 are important for the development of hair follicles and determining hair shape by controlling hair keratin genes.

Oleanolic acid promotes hair growth by increasing cell proliferation through the Wnt/β-catenin pathway.

Increased ODC expression makes normally tumor-resistant mice more prone to tumor development.

HtrA2 activity is crucial for normal hair growth by regulating fat cell development.

Changing the C-ring structure in certain compounds can make them better at blocking a specific human enzyme.

The mutant HR bmh protein affects hair follicle formation by failing to repress vitamin D receptor activity.

High levels of ODC and a mutant Ha-ras gene cause tumors in mice.

The conclusion is that tritium-labeled testosterone metabolites can be made and are better converted into dihydrotestosterone in skin cells than in prostate tissue.

Inhibitors of 11β-HSD1 show potential for treating type 2 diabetes but require more testing for safety and effectiveness.

Masculinization in affected individuals occurs gradually after puberty due to hormone changes.

A mutation in the Soat1 gene causes hair structure defects and other health issues in AKR/J mice.

Mutations in the HEPHL1 gene cause abnormal hair and cognitive issues.

Enzyme purified and characterized for minoxidil sulphation in rat liver.

Hair sensitivity to androgens is partly controlled by specific enzyme expressions in different hair areas.