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Retinoic acid production and signaling in hair follicles are regulated by location and timing, affecting hair growth and cycling.

DHEA stimulates skin oil glands and could help postmenopausal women, with potential for acne and excessive hair growth treatments.

DHT affects hair follicle cells by changing microRNA levels, leading to less cell growth and more cell death.

A new enzyme in rats may help regulate hair growth.

Differences in enzyme activity and androgen receptors in hair follicles may explain why hair loss appears differently in men and women.

New compounds were made that effectively block enzymes related to prostate issues and hair loss.

Aspergillus niger culture creates two finasteride derivatives with enzyme-inhibiting effects.

DHT, not testosterone, mainly affects hair growth and related conditions.

The body's change of testosterone into DHT is not necessary for testosterone's muscle and sexual effects.

Finasteride effectively inhibits the enzyme steroid 5 alpha-reductase II.

The DHT immunoassay is unreliable due to high cross-reactivity with testosterone.

Topical 17 alpha-propylmesterolone increases hair cell growth in androgenetic alopecia.

A genetic marker linked to a type of hair loss was found in most patients studied.

Dihydrotestosterone causes p27 protein breakdown in ovarian and breast cancer cells by binding with SKP2.

Adolescents with PCOS have higher levels of certain androgens, which are linked to hair growth but don't help diagnose PCOS better than testosterone levels.

Human beard hair follicles change testosterone into other related hormones, but don't much change androstenedione into testosterone.

15-lipoxygenase helps keep skin healthy by reducing inflammation.

Dihydrotestosterone and 5α-Reductase play a role in hair loss and prostate health, and finasteride can increase hair growth in men without affecting sperm production, but it doesn't work for postmenopausal women with hair loss.

New compounds were made and tested, with compound 6 showing potential for treating prostate-related diseases.

Smaller substituents at C-17 enhance the inhibitory activity of progesterone derivatives on 5alpha-reductase.

The root extract of Untica mairei Levl may help treat benign prostatic hyperplasia.

Mice without the enzyme HSD17B3 still produce normal testosterone, suggesting they have different ways to make it compared to humans.

Vitamin D receptor can control the hairless gene linked to hair loss even without vitamin D.

Activating ALDH2 can boost hair growth.

The activity of a specific rat enzyme in the prostate and epididymis is highly dependent on the acidity level.

Four patients with a type of rickets and hair loss had different mutations in their vitamin D receptor gene, causing it to not work properly.

New compounds were made that effectively block a specific enzyme related to androgen conditions.

Vitamin C derivative reduces hair loss-related protein in cells.