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A boy with severe Vitamin D-resistant rickets did not respond to treatment and lacked a common symptom, suggesting a need for alternative treatments.

The vitamin D receptor can work without its usual activating molecule.

The hair test for vitamin D could be a useful alternative to blood tests, providing a longer-term vitamin D status, but more research is needed.

Vitamin D levels are not correlated with chronic hair loss in Telogen Effluvium patients.

Skin cells produce and activate vitamin D, which regulates skin functions and supports hair growth.

Measuring 24-OHase induction helps identify defects in vitamin D processing and predict treatment response.

Vitamin D3 affects cell differentiation in specific skin areas.

The enzyme for activating vitamin D is found in many body tissues, not just the kidneys.

The enzyme 25 Hydroxyvitamin D 1 α-Hydroxylase is essential for healthy skin and recovery after skin damage.

Adding a specific gene to skin cells can help treat skin disorders like psoriasis.

Human adrenals and gonads have a unique enzyme for steroid hormone production.

1,25-(OH)2D3 helps hair grow by blocking certain harmful signals.

A specific gene mutation causes vitamin D resistance, but certain vitamin D analogs might help.

Four patients with a type of rickets and hair loss had different mutations in their vitamin D receptor gene, causing it to not work properly.

Vitamin D made by the skin plays a role in immune defense and skin health, and more research is needed to understand its full effects.

A new gene mutation causes vitamin D resistance and rickets, treatable with calcium therapy.

A rare genetic mutation causes resistance to vitamin D, leading to severe rickets and requiring high doses of calcium and vitamin D for treatment.

Vitamin D has potential benefits for cancer prevention, heart health, diabetes, obesity, muscle function, skin health, and immune function, but clinical results are mixed and more research is needed.

A rare genetic mutation causes resistance to vitamin D, leading to severe rickets and requires high doses of calcium and vitamin D for management.

The vitamin D receptor is involved in multiple body functions beyond calcium regulation, including immune response and rapid reactions not related to gene activity.

DDR-II hair follicle cells don't respond to 1, 25-dihydroxyvitamin D3, aiding diagnosis.

Vitamin D and calcium are important for quick and effective skin wound healing.

Women with PCOS have higher hair cortisol levels, which are linked to worse metabolic and inflammatory conditions.

Vitamin D levels in hair don't match those in blood, so hair isn't a reliable indicator of Vitamin D status.

A specific gene mutation causes vitamin D-resistant rickets and hair loss.

New mutations in the VDR gene cause vitamin D-resistant rickets without hair loss.

A girl had rickets due to a gene mutation affecting vitamin D response.

Vitamin D may help regulate cholesterol and influence prostate cancer development.

The vitamin D receptor can regulate genes and affect hair and hormone levels without its usual activator.

Vitamin D receptor binds similarly to natural and synthetic ligands, affecting gene regulation.