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Sinapic acid and glabridin together help hair growth in androgenetic alopecia.

Removing β-catenin in certain stem cells causes hair whitening and pigmentation issues.

Involucrin is a useful marker for keratinocyte differentiation in mice.

Activated LEF/TCF complexes are crucial for hair development and cycling.

The Wave complex controls skin growth by suppressing certain signals.

African spiny mice can regenerate skin, hair, and cartilage, but not muscle, and their unique abilities could be useful for regenerative medicine.

CD4 T cells can cause alopecia areata by activating CD8 T cells to attack hair follicles.

miR-5110 affects alpaca pigmentation by altering specific gene expressions.

Par3–mInsc and Gαi3 work together to ensure proper cell division orientation in skin development.

A new mouse mutation causes skin and hair defects due to a gene change.

Boosting β-catenin signaling in certain skin cells can enhance hair growth.

JAK inhibitors help hair growth by boosting beta-catenin activity in hair root cells.

The BMP2 gene is more active in the early growth phase of Cashmere goat hair and may affect hair regeneration and textile production.

Androgens prevent hair growth by changing Wnt signals in cells.

Injecting monocyto-angiotropin into hare skin increases hair growth by forming new blood vessels.

The HCR gene contributes to psoriasis risk.

Researchers used CRISPR/Cas9 to create a goat with a gene that increased cashmere production by 74.5% without affecting quality.

Gender-affirming hormone therapy affects metabolism differently based on treatment type.

3,4,5-tri-O-caffeoylquinic acid promotes hair growth by activating the β-catenin pathway.

CD44 variant changes start alopecia areata, but don't maintain it.

β-catenin is essential for stem cell activation and proliferation in hair follicles.

CDP is crucial for lung and hair follicle cell development.

The HtrA1L364P mutation causes brain dysfunction and blood vessel damage.

The mutation causes hairless mice due to mislocalized and dysfunctional HR protein.

Botulinum toxin A helped stop hair loss and grow new hair in mice.

Overexpression of LRIG3 in skin causes hair loss.

The TCL1 transgenic mouse model is useful for understanding human B-cell leukemia and testing new treatments.

Blocking β-catenin in skin cells improves hair growth during wound healing.

Gene knockout mice developed scars similar to human hypertrophic scars, useful for studying scar progression.