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Genetically repaired stem cells may treat certain genetic diseases, Th17 cells are key in fighting systemic fungal infections, hair loss in AGA is due to progenitor cell loss, and α-synuclein transfer might contribute to Parkinson's disease progression.

ING5 is crucial for stem cell maintenance and preventing certain cancers.

The method successfully created stable transfection donor cells for goat hair follicle research.

Blocking CXCL12 can reverse hair loss and fibrosis in androgenetic alopecia.

Basement membrane changes are crucial for hair follicle development.

Overexpressing the MSX-2 gene in mice causes skin and hair growth issues.

Harlequin mutant mice have hair loss due to low AIF protein levels and retroviral element activity.

SHISA6 helps maintain certain stem cells in mouse testes by blocking signals that would otherwise cause them to differentiate.

A new microneedle treatment shows promise for better hair regrowth in androgenetic alopecia.

The OVOL1 gene, controlled by β-catenin, is crucial for creating hair follicles.

Adult spiny mice recover better from heart attacks than common lab mice.

Wnt/β-catenin activators can help regrow hair lost due to diabetes.

M-CSF-stimulated myeloid cells can cause alopecia areata in mice.

A mutation in the Soat1 gene causes hair structure defects and other health issues in AKR/J mice.

Dermal lymphatic vessels help hair growth by affecting hair cycle phases.

Activated protein C helps protect mice from long-term radiation damage.

Bone Morphogenetic Proteins (BMPs) are important for hair growth, and their decrease due to hormones could lead to hair loss, but adding more BMPs could promote hair growth.

Agouti protein affects melanocortin receptors through competitive antagonism and receptor down-regulation.

Progranulin overexpression leads to shorter, thinner hair and increased cell death in mouse hair follicles.

Too much parathyroid hormone-related protein in skin disrupts hair growth in mice.

Male hormones control a specific gene in hamster skin, with different hormones having varying effects.

The study maps how genes are regulated during mouse hair growth.

Too much β-catenin activity can mess up the development of mammary glands and make them more like hair follicles.

Stress can cause a type of hair loss in mice lacking the CCHCR1 gene.

Blocking TGFβ can help treat fibrotic skin conditions by promoting fat cell formation.

The new Hairless R/J mice model improves imaging for tumor monitoring and cancer therapy evaluation.

Loss of Pten in certain hair follicle stem cells increases skin cancer risk.

CD61 is important for mouse tooth cell growth and works through Lgr5.