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Mice with too much sPLA₂-IIA have hair loss and poor wound healing due to abnormal hair growth and stem cell depletion.

Scientists restored fertility in male mice lacking a key fertility gene by using a modified gene.

The improved genome of the African spiny mouse will help understand its tissue regeneration abilities.

A new mouse mutation causes skin and hair defects due to a gene change.

AP-1 controls tumor cell type by affecting key signaling pathways.

Early involution in Hirosaki hairless rats' mammary glands is linked to a unique modification of STAT5A.

Overexpressing a specific enzyme in mice causes hair loss and female infertility.

The improved genome of the African spiny mouse helps study its tissue regeneration.

Aire mutation reduces alopecia areata, while Notch4 mutation prevents it in mice.

The UHS promoter is specific to mouse hair follicles.

High levels of ODC and a mutant Ha-ras gene cause tumors in mice.

Researchers created a new mouse model for studying Citrullinemia Type I and similar conditions, showing symptoms and treatment responses like those in humans.

TGFα gene mutation in mice causes abnormal skin, wavy hair, curly whiskers, and sometimes eye inflammation.

Altering SSAT affects fat metabolism and body fat in mice.

Overexpression of sPLA2-IIA in mouse skin reduces hair stem cells and increases cell differentiation through JNK/c-Jun pathway activation.

Krt6a-Cre transgenic mice help study gene effects on hair follicle development and tumor suppression.

The study showed that a specific DNA sequence can control gene expression in hair growth areas of mice.

The study developed a mouse model for Alopecia Areata that responds to treatment, useful for future research.

TNF alpha in skin cells causes weight loss, hair and fat issues, and skin inflammation in mice.

Activating the sonic hedgehog pathway in chicken embryos can permanently change scales to feathers.

Alopecia areata in these mice is inherited, more common in young females, and can be treated with triamcinolone acetonide.

Scientists made a mouse that can be made to lose hair and then grow it back.

A mouse model for studying scleroderma in chronic graft-versus-host disease was successfully created.

Activated β-catenin affects hair growth and skin thickness, and changes are reversible.

Researchers developed a mouse model that tracks hair growth using bioluminescence, improving accuracy in studying hair cycles.

Overexpressing 14-3-3σ in mice skin reduces cell growth and hair density.

Increased ODC expression makes normally tumor-resistant mice more prone to tumor development.

Increasing COX-2 in mouse skin causes bigger sebaceous glands and thinner hair, but stopping COX-2 can reverse hair thinning.

Activating Tgr5 may help treat hair loss and bone loss.