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Protein kinase C epsilon may promote skin cancer development after UV exposure by affecting nearby cells.

Ca²⁺-mediated protein citrullination controls cell growth in the CNS and may help treat brain tumors.

Noggin promotes skin tumors by activating Wnt and Shh pathways.

PTCH gene mutations contribute to basal cell carcinoma development.

The Gsdma3 gene is essential for normal hair development in mice.

Scube3 gene affects mouse embryo growth in multiple areas, but needs more research.

Overexpressing PKCα in mice skin increases inflammation but doesn't affect tumor growth.

Cathepsin L is essential for normal hair growth and development.

ATP-sensitive potassium channels are important for hair growth.

Basal cell carcinoma with matrical differentiation is a rare type linked to hair follicles, with .-catenin important for its development.

The OVOL1 gene, controlled by β-catenin, is crucial for creating hair follicles.

The system allows precise control of gene expression in mouse skin, useful for studying skin biology.

Protein kinase C epsilon may increase skin cancer risk by affecting nearby cells.

The Hedgehog signaling pathway is linked to cancer development, and targeting it with inhibitors shows promise but faces challenges like resistance.

Tsc2-deficient stem cells can help understand and treat TSC-related tumors.

Involucrin gene expression is controlled by specific proteins and signaling pathways.

Deleting the CD271 gene in mouse skin cells leads to disorganized skin and increased hair growth, suggesting CD271 is important for skin health.

TSC2 is crucial for proper hair follicle development and patterning.

Wnt signaling controls whether hair follicle stem cells stay inactive or regenerate hair.

The Ysc84/SH3yl1 protein family is important for cell movement and the process of taking in materials by interacting with actin and cell membranes.

Activin B improves wound healing and hair growth by helping stem cells move using certain cell signals.

Lack of Ctip2 in skin cells delays wound healing and disrupts hair follicle stem cell markers in mice.

EGFR helps hair follicles transition properly by controlling Stathmin levels.

SHISA6 helps maintain certain stem cells in mouse testes by blocking signals that would otherwise cause them to differentiate.

Both main and alternative Wnt signaling are important for regrowing rodent whisker follicles.

cgVEGF164 boosts hair follicle growth in mice.

Tenascin-C and tenascin-W help control stem cell movement and growth in whisker follicles.

Certain compounds can boost hair and nail growth.