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Ritlecitinib effectively reduces hair loss in JAK inhibitor-naïve alopecia areata patients.

Ritlecitinib was generally well tolerated in children with alopecia areata.

Finasteride helps female-pattern hair loss.

Mouse hair keratins mHa1 and mHb4 can't form a strong network on their own in cells.

Mutant MK6a transgenes in mice cause blistering, hair loss, and potential human alopecia.

CS12192 effectively treats alopecia areata with better safety than current options.

Sacituzumab tirumotecan shows promise in treating breast cancer with manageable side effects.

Keratoacanthoma is a common, usually non-dangerous skin tumor that looks like squamous cell carcinoma but rarely becomes severe.

Tofacitinib helped regrow hair in patients with alopecia, with few side effects.

Trichohyalin in hair can trigger immune attacks in alopecia areata.

Expanded CD8+ T cells are linked to Alopecia Areata and may cause relapse after treatment.

Removing Mediator 1 causes teeth cells to turn into hair cells.

The AVET system effectively delivers genes to human keratinocytes and may help treat skin diseases.

TH antibodies in vitiligo and AA patients recognize the same protein parts.

TG5 helps maintain hair follicle health, while TG3 aids in hair shaft development.

Tofacitinib can regrow hair in alopecia areata patients, but some may experience flares during treatment.

Targeting mitophagy may help treat alopecia areata by reducing inflammasome activation.

Foxn1 helps skin cells mature by controlling a specific protein's activity.

TGF-β1 from dermal papilla cells suppresses hair growth, and targeting it may help treat androgenetic alopecia.

CTHRC1 helps sweat glands recover by rebuilding nearby blood vessels.

Upadacitinib effectively improved severe atopic dermatitis and alopecia universalis in a 29-year-old man.

A KRT32 gene variant causes loose anagen hair syndrome.

Plet-1 protein helps hair follicle cells move and stick to tissues.

IFN-γ production by CD4 T cells is crucial for causing alopecia areata.

Blocking α1-integrin makes adult fibroblasts more like foetal ones, improving their movement and aiding wound healing.

Newly designed proteins can effectively degrade specific proteins in cells, offering a potential new therapy method.

ADM scaffolds help skin heal by promoting a healing-type immune response.

Retinoic acids temporarily change skin stem cells to a slower cycle and more differentiated state.