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Stem cells from elderly skin can become neurons, offering potential for brain therapy.

Keratinocytes help manage skin nitrogen metabolism, varying by age and skin area.

Pitx2 helps outer root sheath cells differentiate but can't start hair growth on its own.

The mouse hairy ears mutation causes longer ear hair due to changes in gene expression.

Injecting monocyto-angiotropin into hare skin increases hair growth by forming new blood vessels.

Trichohyalin, a hair follicle protein, has a part with repeating patterns of 23 amino acids.

Overexpression of Hoxc13 in hair cells causes hair loss and skin issues.

Modulating the BTNL2 pathway can prevent hair loss in mice.

WNT10A is important for tissue development and linked to various human disorders.

TMPRSS6 is crucial for controlling hepcidin and normal iron absorption.

Mutations in the hHb6 gene cause the hair disorder monilethrix.

Mice with a changed Hr gene lose and regrow hair due to changes in the gene's activity.

HAT-L4 is crucial for preventing body fluid loss by maintaining skin barrier integrity.

Certain genetic variants reduce enzyme activity, contributing to non-classic congenital adrenal hyperplasia.

The MtAnn3 gene affects root hair growth and is influenced by cytokinin.

FOXN1 gene variants cause low T cells and immune issues from birth.

AXR3 and SHY2 genes control the growth and timing of root hair development in plants.

Disrupting Acvr1b in mice causes severe hair loss and thicker skin.

AH-001 could be a safer and more effective treatment for hair loss.

The new model helps understand and develop treatments for genetic skin disorders like AEC.

A new mouse hair mutation, called hague, is semidominant and unstable, but the exact cause is unknown.

Truncated hHb1 keratin may play a role in breast cancer cell transformation.

The nude gene is important for skin and hair development.

Type I keratin genes are closely linked to the rex locus on mouse chromosome 11, affecting hair development.

Lnc056 helps hair follicle stem cells grow by increasing TRIP6 expression.

Trichohyalin is modified by enzymes to form strong structures in hair cells.

A new genetic variant causes trichothiodystrophy in two brothers, but their mother may carry it without showing symptoms.