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A new compound, HTPI, promotes hair growth by protecting cells from damage and regulating energy use.

Too much keratin 16 in mice skin causes abnormal skin thickening and structure.

IL-9 increases skin cell movement but decreases their ability to invade, and this effect is controlled by cell contractility, not by MMPs.

Naked-mouse hair lacks certain proteins and has less soluble fibril.

Overactivating Hedgehog signaling makes hair follicle cells in mice grow hair faster and create more follicles.

Keratin's mechanical properties are influenced by hydrogen bonds and secondary structure, and can be improved with the SPD-2 peptide.

New mutations in the hairless gene may cause hair loss and affect bone development.

Blocking IL-12 can help treat alopecia areata by preventing hair follicle immune issues.

Certain skin proteins can form anchoring structures without the protein AMACO.

The hairless (hr) gene is essential for normal hair follicle function and its mutation leads to hair loss.

Dormant melanoma cells in mice interact minimally with memory T cells due to a suppressive tumor environment.

Mutant MK6a transgenes in mice cause blistering, hair loss, and potential human alopecia.

COX2 and ATP synthase control the size of hedgehog spines.

A substance called DKK-1 increases in balding areas and causes hair cells to die when exposed to DHT.

A mutation in the human hairless gene causes alopecia universalis.

The Notch signaling pathway helps in mouse hair development through a noncanonical mechanism that does not rely on RBPj or transcription.

Hedgehog signaling can create new hair follicles but may also cause tumors.

Gab1 protein is crucial for hair growth and stem cell renewal, and Mapk signaling helps maintain these processes.

LRIG1 protein affects hair growth by regulating skin receptors, leading to hair loss when overexpressed.

Type II keratins are uniquely phosphorylated during stress and mitosis, affecting their structure and function.

Mutations in certain receptors can cause diseases and offer new treatment options.

Altering the keratin 17 gene in mice hair follicles caused temporary hair issues, but changes were minimal and short-lived.

AtZP1 protein stops root hair growth in plants by blocking certain genes.

ODC overexpression in hair cells increases tumor growth by reducing Notch signaling.

Trichocyte filaments have a low-density core and may include proteins for hair structure.

A mutation in the KRT25 gene causes woolly hair and hair loss.