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Leptin-deficient mice, used as a model for Type 2 Diabetes, have delayed wound healing due to impaired contraction and other dysfunctional cellular responses.

Cytarabine can cause multiple organ toxicities, especially neurotoxicity, but better research methods are needed to fully understand and predict these effects.

Stem cells slowed lung tumor growth but increased colon tumor growth in mice.

Porcine ADM scaffold helps hair growth in mice.

Overexpressing follistatin in mice delays wound healing and reduces scar size.

Overexpressing a specific enzyme in mice causes hair loss and female infertility.

Cannabinoids may help treat various skin conditions.

DNA virus-induced tumors have consistent isozyme profiles, unlike other tumor types.

Using both bone marrow concentrate and platelet-rich plasma together improves skin wound healing in mice.

Understanding genetic variations in mice is crucial for studying skin, hair, or nail abnormalities.

A gene mutation worsens skin irritation in mice due to a lack of certain fats.

IL-1 receptor absence in mice leads to skin cysts and changes in immune response after UVB exposure.

Mice with the ab gene have abnormal sebaceous gland development, affecting sebum production.

Zebularine improved skin structure but delayed wound healing in diabetic mice.

Ovalbumin–aluminum sensitization causes increased pain sensitivity and nerve changes in mice.

Luteolin can reduce hair graying in mice, with external treatment being more effective.

Mice with more Flightless I protein grew back their claws better after amputation.

Eclipta alba extract was found to be effective in promoting hair growth in hairless mice.

Th22 cells are essential for Tβ15-induced hair growth in mice.

Implanting hair-follicle stem cells in mice brains helped repair brain bleeding and reduced brain inflammation.

Fermented mushroom extract reduces intestinal inflammation in mice.

GCN reduces lung inflammation and damage from air pollution in mice.

Radiation significantly slows down wound healing in mice.

Cathepsin L deficiency causes hair and skin issues in mice.

The gene HDC is important for the development of hair follicles in newborn mice.

The Itpr3 gene causes a specific hair pattern in mice.

The E2 protein affects gene activity in hair follicles of mice.

A new model for hair regeneration in mice was created in 2015, which is faster and less invasive than the old method, producing normal hairs in about 21 days.

Blocking DGAT1 reduces oil gland size in mice and dogs, but only mice experience hair loss.

The ACTH/MC2R system is crucial for controlling hair growth cycles in mice.