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The human scalp has different types of pigment cells in hair follicles with varying abilities to produce pigment.

Rushton's hyaline bodies form from hair keratin and blood substances.

Myeloid cells can turn into skin and hair cells to help heal wounds.

Seven genes are highly expressed in both germ-line and hematopoietic stem cells.

Canine hair follicles contain stem-like cells with high growth potential.

Tph cells are linked to the severity of systemic lupus erythematosus.

Innate lymphoid cells type 1 may contribute to alopecia areata.

Monoclonal antibodies LT-1, LT-2, and LT-7 help diagnose certain blood cancers.

The antibody created from BCC tissues reacts similarly to both BCC and hair follicles, suggesting BCC may come from hair follicle cells.

Neutropenic patients show significant changes in immune cell types and lower neutrophil and natural killer cell percentages.

ILC1-like cells can cause alopecia areata by affecting hair follicles.

Berberine may reduce prostate enlargement by blocking inflammation.

Thymosin beta 4 protects cells from damage by blocking a harmful microRNA and boosting a protective gene.

Canine hair follicles have stem cells similar to human hair follicles, useful for studying hair disorders.

Schwann cell and M2 macrophage interactions contribute to keloid growth by increasing matrix deposition.

CD34-positive cells help repair and form new blood vessels in salivary glands after radiation.

Hair follicle cells resist turning into skin cells.

People with atopic dermatitis have more CD4+ T cells that respond to a certain bacterial lipid, which may play a role in the skin condition's inflammation.

People with alopecia areata have fewer IL-10 producing immune cells, which might contribute to the condition.

Mouse skin has special cells in the epidermis that decrease with age and are linked to keratinocyte stem cells.

Mast cells and the CB1 receptor may be key in causing IgA vasculitis.

The research created a detailed map of skin cells, showing that certain cells in basal cell carcinoma may come from hair follicles and could help the cancer grow.

BST2 is a key marker for hair loss disease alopecia areata.

Fibroblast and immune cell interactions affect tissue repair and fibrosis.

CD34 is absent in most basal cell carcinoma cells but present in surrounding skin.

Mast cells may significantly contribute to central centrifugal cicatricial alopecia.

Arg1+ macrophages may play a role in causing alopecia areata.

Ancient immune and signaling pathways still regulate blood cell development.

Natural killer cells play a key role in autoimmune skin diseases like pemphigus vulgaris.