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Mice genetically modified to produce more CD109 in their skin had less inflammation and better healing with less scarring.

Spiny mice can regenerate tissues instead of forming scars.

Certain proteins, caspases-1 and -11, are important in the early development of skin inflammation in mice.

SHISA6 helps maintain certain stem cells in mouse testes by blocking signals that would otherwise cause them to differentiate.

NG2 is crucial for normal skin and hair development in mice.

The Walleye dermal sarcoma virus cyclin causes excessive skin cell growth in mice.

Overactivating Hedgehog signaling makes hair follicle cells in mice grow hair faster and create more follicles.

Overexpressing ovine β-catenin in mice skin increases hair follicle density and growth.

Gorab deficiency speeds up skin aging by disrupting protein regulation and reducing collagen.

MPZL3 protein helps control the size of oil glands and the growth of oil-producing cells in both mice and humans.

Lower GATA3 levels in mice help hair regrow by changing certain immune cells.

Androgen loss may speed up hair greying.

Age and androgen levels significantly affect meibomian gland structure and function.

Alopecia areata involves specific immune cells, offering potential treatment targets.

Mutations in the POC1A gene can cause a unique form of extreme insulin resistance and short stature.

Activin B helps start and grow hair follicles in mice.

Lack of Evi in skin causes psoriasis-like symptoms in mice.

A small BOTOX dose improved sperm production and movement in older mice.

Iron deficiency in mothers causes hair loss in their baby mice.

Alopecia in VDR knockout mice is due to a defect in hair cycle initiation, not keratinocyte issues.

Rac1 is vital for hair follicle health but not needed for skin maintenance.

TGFBR1 slows down cell growth in fine-wool sheep hair follicles.

Amino acid storage proteins are essential for maintaining stem cells in female fruit flies.

Selenium imbalance can cause hair loss and skin issues.

Skin lesions in Carney Complex are caused by a gene change in some skin cells that leads to increased pigmentation and may lead to tumors.

Bendamustine combined with tucidinostat may effectively treat adult T-cell leukemia.