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Platelet-rich fibrin may help reduce nonmelanoma skin cancer cell growth.

Genetic variants in specific genes cause central centrifugal cicatricial alopecia.

A new gene location for Keratosis follicularis squamosa was found on chromosome 7p14.3-7p12.1.

Activated protein C helps protect mice from long-term radiation damage.

SLC24A5 shows a clear selective sweep, but no link to UV radiation intensity.

Certain genetic variants in keratins increase the risk of tooth decay.

A boy with Oculodentodigital syndrome had a unique GJA1 gene mutation causing his symptoms.

TSC2-/meth cells can cause skin lesions, hair growth, and lung issues, and may be treated with chromatin remodeling agents.

Hairless gene not strongly linked to baldness.

Damaged hair follicles make mice more prone to skin inflammation and skin cancer after UV exposure.

Androgen receptors are key for development and health, affecting conditions like prostate cancer and male pattern baldness.

Understanding where cancer cells come from helps create better prevention and treatment methods.

An 8-year-old girl has a rare, irreversible hair loss condition caused by a genetic mutation.

The TCL1 transgenic mouse model is useful for understanding human B-cell leukemia and testing new treatments.

The research suggests that autophagy-related genes might play a role in causing alopecia areata.

Retinoic acid affects skin and hair health by working with specific receptors, and its absence can lead to hair loss and skin changes.

Canagliflozin helps overcome progestin resistance in certain endometrial cancer cells.

Androgen receptor signaling causes early aging of cells important for hair growth by damaging their DNA.

A new genetic mutation in the CAST gene may link PLACK syndrome to alopecia areata.

Trichilemmal carcinoma shares genetic traits with other skin cancers, suggesting similar treatment options.

Certain genes and X chromosome patterns may significantly contribute to the development of hair loss.

EBV infection increases a specific keratin variant in carcinoma cells, possibly affecting cell structure and cancer progression.

Two different mutations in the vitamin D receptor gene cause different symptoms and responses to treatment in Lebanese patients with hereditary rickets.

A rare genetic mutation causes Woodhouse-Sakati syndrome symptoms.

Cronkhite-Canada syndrome may be more treatable and less severe than previously thought.

RAGE is a potential target for melanoma treatment, but its effectiveness is uncertain due to variable expression levels.

Kennedy's disease leads to muscle weakness and sensory issues, has no cure but manageable symptoms, and future treatments look promising.

Collagen XVII is important for cell functions and its absence can worsen cancer outcomes.

Trichilemmal carcinoma shares genetic traits with other skin cancers, suggesting similar treatment options.