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Removing myelin protein zero-like 3 in mice leads to better metabolism and resistance to obesity.

Mice with human fetal thymic tissue and stem cells developed symptoms similar to chronic graft-versus-host disease.

The N gene affects the protein makeup of mouse hair.

Adult mice with CBS deficiency show minimal health issues and normal lifespan despite high homocysteine levels.

Use ethical and humane practices in mouse research.

The lanceolate hair-J mutation in mice helps understand human hair disorders like Netherton's syndrome.

Homozygous K5Cre transgenic mice have wavy hair and faster cancer progression.

Older mice healed wounds better but lost more weight and might have weaker immune systems afterward.

K6 gene expression can be controlled and manipulated in mice for studying skin disorders.

A specific gene region can control targeted and responsive gene expression in mice, useful for skin disorder treatments.

A gene mutation in mice causes skin defects and early death.

Li2CO3 improved skin disease in a mouse model of Focal Dermal Hypoplasia without toxicity.

Mice with too much Claudin-6 have skin barrier problems and abnormal hair growth.

Mice without the IL-6 gene had more hair growth after injury due to higher activity of a related protein, Stat3.

Noggin gene inactivation causes skeletal defects in mice, varying by genetic background.

Hairless mice are more vulnerable to Listeria infection, but gut microbiota can help reduce damage.

Giving immune serum from vaccinated mice to mice without T cells prevents infection and tumor growth.

Mouse keratin 6 isoforms have different expression patterns in various tissues.

The model using human skin on mice helps study human sebaceous glands.

Mice with human-like EGFR had growth issues, skin defects, heart problems, and unusual bone development.

Researchers created a mouse model for Cushing's syndrome to study glucocorticoid excess and potential treatments.

Genetic mutation and carcinogen treatment are both needed for skin cancer to develop in these specific mice.

Male-to-female skin grafts in mice are rejected due to sex-linked antigens.

Krt6a-Cre transgenic mice help study gene effects on hair follicle development and tumor suppression.

Mouse keratin 6 genes evolved independently from human ones and are regulated differently.

Damaged hair follicles make mice more prone to skin inflammation and skin cancer after UV exposure.

A new mouse model for vitiligo helps study immune responses and potential treatments.

External factors can cause skin cancer cells that usually don't spread to grow and form tumors in mice.

The improved genome of the African spiny mouse will help understand its tissue regeneration abilities.