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Melanocyte pathology requires keratinocyte hyperplasia and regulation dysfunction.

Activating β-catenin increases melanocytes and decreases Schwann cells.

Introducing the OTC gene improved symptoms in mice with OTC deficiency.

The mTurq2-Col4a1 mouse model shows how the basement membrane develops in live mammals.

CDP is crucial for lung and hair follicle cell development.

Vitamin D receptor knockout mice have significant motor impairments but no cognitive deficits.

Removing β-catenin in certain stem cells causes hair whitening and pigmentation issues.

A gene mutation in mice causes hair loss, weak bones, and protein buildup, showing how protein processing issues can lead to diseases.

Blocking BACE1 and BACE2 enzymes causes hair color loss in mice.

Mice without the p21 gene can fully regenerate injured ears due to reduced Sdf1 increase and leukocyte recruitment, suggesting new ways to induce tissue regeneration in mammals.

Spiny mice have resilient, large mitochondria that help them regenerate tissue.

Lactococcus lactis strain H61 may reduce some ageing symptoms.

Foxn1 is essential for hair pigmentation by directing pigment transfer to hair cells.

Spiny mice are better at regenerating hair after injury than laboratory mice and could help us understand how to improve human skin repair.

The balance between cell renewal and differentiation controls the growth of cancerous cells in mouse skin.

The HCR gene contributes to psoriasis risk.

SMR-2 and SMR-6 are much more toxic than retinoic acid, causing severe symptoms and organ damage in mice.

Acid inside cells speeds up aging and turns on aging signs in mice.

Gonadal hormones significantly affect the severity of alopecia areata in mice.

ING5 is crucial for stem cell maintenance and preventing certain cancers.

Overexpressing ovine β-catenin in mice skin increases hair follicle density and growth.

The hairless gene in Kunming mice is important for hair and skin, and shows genetic variations.

CD8+ T cells attack hair follicle stem cells, causing scarring and hair loss.

A mutation in the Sgkl gene causes defective hair growth in mice.

CD8+ T cells expand significantly in alopecia areata, suggesting new treatment targets.

SMR-2 and SMR-6 are much more toxic than retinoic acid, causing severe side effects.

Spiny mice regenerate skin better than laboratory mice due to larger hair bulges, more stem cells, and different collagen ratios.

Mice with human skin protein K8 had more skin problems and cancer.

Overexpressing NSD3 in mice causes breast cancer-like tumors and gland abnormalities.