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Spiny mice are better at regenerating hair after injury than laboratory mice and could help us understand how to improve human skin repair.

Male and female mice handle stress differently.

The balance between cell renewal and differentiation controls the growth of cancerous cells in mouse skin.

The HCR gene contributes to psoriasis risk.

SMR-2 and SMR-6 are much more toxic than retinoic acid, causing severe symptoms and organ damage in mice.

Acid inside cells speeds up aging and turns on aging signs in mice.

Gonadal hormones significantly affect the severity of alopecia areata in mice.

ING5 is crucial for stem cell maintenance and preventing certain cancers.

Overexpressing ovine β-catenin in mice skin increases hair follicle density and growth.

The hairless gene in Kunming mice is important for hair and skin, and shows genetic variations.

CD8+ T cells attack hair follicle stem cells, causing scarring and hair loss.

A mutation in the Sgkl gene causes defective hair growth in mice.

CD8+ T cells expand significantly in alopecia areata, suggesting new treatment targets.

SMR-2 and SMR-6 are much more toxic than retinoic acid, causing severe side effects.

Spiny mice regenerate skin better than laboratory mice due to larger hair bulges, more stem cells, and different collagen ratios.

Mice with human skin protein K8 had more skin problems and cancer.

Injecting newborn mice with a niacin blocker caused skin, gut, and brain damage similar to human pellagra.

Overexpressing NSD3 in mice causes breast cancer-like tumors and gland abnormalities.

Human cathepsin V can replace mouse cathepsin L to maintain normal skin and hair in mice.

A new mutation in the hairless gene causes hair loss and skin wrinkling in mice.

Alopecia areata is an autoimmune disease that targets hair follicles.

CD61 is important for mouse tooth cell growth and works through Lgr5.

Mice with LSS deficiency showed hair loss and cataracts, similar to humans, and can help in understanding and treating this condition.

Two genes, ERBB4 and ROR1, may cause the unique pigmentation in Lanping black-boned sheep.

Researchers created a new mouse model, G4, that mimics human PCOS symptoms and links the condition to a specific gene.

Lgr5 and the vitamin D receptor are key in controlling skin inflammation and tumor risk in mice.

A new mouse mutation causes skin and hair defects due to a gene change.

A new mutation in the Hr gene causes hair loss in mice, similar to a human hair disorder.

Transplanted whisker follicles caused hair growth on the spine of mice.

The frizzy mouse and hairless rat mutations are due to changes in the Prss8 gene.