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Changing protein kinase levels in pituitary cells affects calcium flow and beta-endorphin release.

Peripheral nerves are essential for skin wound healing and future treatments could improve by focusing on nerve and blood vessel regeneration.

Intravenous calcium therapy is a safe and effective treatment for a rare type of rickets.

Calretinin identifies the companion cell layer in human hair follicles.

Rat vibrissae structure relates to their sensory function.

Long-term use of seizure medications can disrupt calcium metabolism, but this can be treated with vitamin D or UV light and does not affect seizure control.

CENPV, a new partner of CYLD, helps regulate ciliary acetylated tubulin and is overexpressed in certain skin tumors.

Cinacalcet may help treat hereditary vitamin D resistant rickets safely.

Hair follicles could help develop eye treatments by studying immune responses.

Increased scalp sweating in frontal fibrosing alopecia may be linked to local skin inflammation.

Activating TRPV1 can boost hair growth by involving neurons, macrophages, and fibroblasts.

Engineered nanovesicles from hair follicle stem cells enable scarless healing of infected wounds.

Botulinum toxin type A did not help regrow hair in severe alopecia cases.

Nocturnal calcium infusions improved a boy's severe rickets without causing hair loss.

A new method efficiently isolates and cultures rat hair follicle cells.

Lampreys have a functional vitamin D receptor that may help detoxify harmful substances.

Ivacaftor can protect against noise-induced hearing loss by reducing oxidative stress.

Testosterone increases acidification in the vas deferens by up-regulating V-ATPase.

Genetically modified rats help reveal how vitamin D affects bone and skin health.

The ointment is a safe, effective, and well-tolerated treatment for certain types of psoriasis.

Rat vibrissa follicles are useful for studying hair growth cycles, especially the transition from pro-anagen to anagen.

CaV1.2 helps activate hair follicle stem cells without calcium flux.

Tunisian children with hereditary vitamin D-resistant rickets showed improvement with calcium treatment, and new genetic mutations were identified.

Excess vitamin A causes lasting gland changes in mouse hair follicles.

We now understand more about what causes acne and this could lead to better, more personalized treatments.

Alopecia areata is an autoimmune disease causing hair loss, treatable with immune-modulating drugs, and linked to genetics.

Neurotrophins regulate nerve growth by balancing promotion and suppression.

Nerve growth in mouse skin and hair follicles happens in stages and is closely linked to hair development.