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SETDB1 is essential for controlling DNA methylation, silencing retrotransposons, and maintaining skin cell health, with its absence leading to skin inflammation and hair loss.

Stress can cause a type of hair loss in mice lacking the CCHCR1 gene.

Pigs without the Hairless gene showed skin and thymus changes, useful for studying human hair disorders.

Suppressing ODC activity reduces tumor growth in hair follicles.

Lack of functional CYLD in mice leads to early aging and cancer.

Circ 0020938 slows down hair growth in cashmere goats.

Id2 gene helps keep hair follicle stem cells inactive.

The tumor suppressor gene FLCN affects mitochondrial function and energy use in cells.

Controlled delivery of specific RNA and IL-4 restored hair growth in mice with autoimmune alopecia.

JAK/STAT1 pathway causes hair loss during chemotherapy by reducing Shh in hair follicles.

Scientists created cell lines to study a genetic skin disorder using CRISPR technology.

The protein CCN2 controls hair growth by affecting hair follicle formation and stem cell activity in mice.

Overexpressing 14-3-3σ in mice skin reduces cell growth and hair density.

The timing of when the gene Bmal1 is active affects aging and survival, with its absence during development, not adulthood, leading to premature aging.

NKIRAS2 can suppress certain skin tumors but its effect on cancer varies with context and expression level.

Ablative fractional laser treatment nearly matches the gene reduction effects of topical vismodegib in skin cancer.

Targeting NCoR1 can help treat heart enlargement and dysfunction.

Knocking out certain genes in mice helps understand skin and hair growth problems.

CRISPR/Cas9 and prime editing can potentially fix skin disorder genes safely and effectively.

BMI1 is essential for preventing hair greying and maintaining hair color.

PBX1 helps reduce aging and cell death in hair follicle stem cells by decreasing DNA damage, not by improving DNA repair.

Removing β-catenin in certain stem cells causes hair whitening and pigmentation issues.

A specific microRNA, chi-miR-30b-5p, slows down the growth of hair-related cells by affecting the CaMKIIδ gene in cashmere goats.

CRISPR gene editing reduces harmful molecules in cells from Emery–Dreifuss Muscular Dystrophy patients.

The treatments stopped hair regrowth in mice.

Small molecules KY19382 and KY19334 may help treat skin cancer by reducing CDK1 levels and blocking harmful cell signals.

The protein folliculin, involved in a rare disease, works with another protein to control how cells stick together and their organization, and changes in this interaction can lead to disease symptoms.

The system allows precise control of gene expression in mouse skin, useful for studying skin biology.